ChayanM/MIMIC-Impression-Dataset · Datasets at Hugging Face

File_Path stringlengths 94 94	Impression stringlengths 1 1.56k
MIMIC-CXR-JPG/2.0.0/files/p11548527/s59875528/2dad832e-0a053633-f36d0657-e7435ed1-bd4b2d30.jpg	no evidence of acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p12152814/s54480700/148238aa-4c54523f-b20d800e-b6bdf00a-5b837e43.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p18299853/s56084599/a16153b3-fc53e8c8-3612a34d-3e832b50-1bf81f7e.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p19284781/s59804824/b629f3c2-8417e87d-6f9ace74-51e9b6af-e47382cb.jpg	the left-sided chest tube has been removed. no pneumothoraces are seen. there is volume loss and loculated pleural fluid along the left lateral chest, unchanged. right lung appears well aerated.
MIMIC-CXR-JPG/2.0.0/files/p17255135/s51518702/044ed8c8-ad454c40-6a32494a-fe636754-7eb3ddb8.jpg	no evidence of interstitial prominence. no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p19245983/s54164846/9113059a-7f7d2278-1cf93415-c8add273-78e36ba7.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15250844/s53486242/248e16e4-58620659-7cf82349-bcb7b83c-add305c6.jpg	left basilar atelectasis.
MIMIC-CXR-JPG/2.0.0/files/p17190208/s56630818/1ec4c930-8ab5ca96-7b164d4b-e3da2a50-583c0d8a.jpg	bilateral lower lobe consolidation is unchanged since earlier in the day consistent with persistent pneumonia. upper lungs are clear. there is no pulmonary edema. pleural effusions a small if any. heart size top-normal unchanged. et tube in standard placement. nasogastric drainage tube passes into the stomach and out of view. left pic line ends in the low svc. right jugular line ends in the upper svc. ventriculoperitoneal shunt traverses the right neck chest and upper abdomen common passing out of view. incidental note is made of a large granulomatous lymph node calcifications in the mediastinum and left hilus and a smaller calcification in the left lung.
MIMIC-CXR-JPG/2.0.0/files/p15472839/s51950800/4b5c26d0-1d8a7252-29a94956-8c794186-ae72c3f2.jpg	no radiographic evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p14005529/s53328303/23860e18-863b8fd2-5c2c43fd-f7682230-d968ecba.jpg	normal. no parenchymal abnormality.
MIMIC-CXR-JPG/2.0.0/files/p11869585/s58573724/be6f3b9d-39cf1579-9f4f6eb6-74109ad1-d0a330d4.jpg	in comparison with the study of , the endotracheal tube and nasogastric tube have been removed. the right ij catheter is unchanged. there is again a large right pleural effusion with compressive basilar atelectasis as well as volume loss in the left lower lobe with smaller effusion. the left perihilar opacity is unchanged.
MIMIC-CXR-JPG/2.0.0/files/p19941474/s56724874/9eddb8b2-2d794f3e-8edee33c-7fb2086e-94008db1.jpg	overall, appearances are unchanged compared to the prior study. persistent pneumoperitoneum and loculated left basal pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p18027598/s58932254/d27cf3be-71977e8b-5cc137db-6026a5e4-65ccf8b7.jpg	new retrocardiac opacity on the lateral view potentially due to atelectasis however it was not seen on prior film with similar inspiratory effort, consolidation would also be possible. please correlate clinically.
MIMIC-CXR-JPG/2.0.0/files/p11951640/s51163578/23b2534e-0125166e-4b0286a5-79b37c0d-ea24aa7e.jpg	no radiographic evidence for acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p19737741/s57411352/5b5f5e13-a1379760-07cf6171-a7d4f5f9-5337f217.jpg	the heart remains enlarged. mediastinal contours are unremarkable. the aorta is unfolded and tortuous. lungs are well inflated without evidence of focal airspace consolidation to suggest pneumonia. no pleural effusions or pneumothorax. no evidence of pulmonary edema. mild eventration of the right hemidiaphragm.
MIMIC-CXR-JPG/2.0.0/files/p15245907/s58995740/937dbe36-5166e05c-c5c42194-ad585161-cbe75187.jpg	there continues to be a layering left effusion with associated airspace opacity which likely reflects atelectasis. there is likely a smaller right effusion with linear and patchy opacities at the base also suggestive of atelectasis. possible minimal to mild interstitial edema. overall cardiac and mediastinal contours are likely stable although difficult to assess due to the patient rotation on the current study. no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p13485392/s59507835/8b31cebf-37df1488-d63ed378-8e17c8b3-35d8cf6c.jpg	as compared to radiograph, widespread pulmonary opacities have apparently progressed and likely reflect worsening acute lung disease process such as infectious pneumonia with possible associated ards superimposed upon chronic interstitial lung disease evident on prior outside ct of. no other relevant changes since recent study.
MIMIC-CXR-JPG/2.0.0/files/p11662094/s54199948/71f651ca-c5961a72-06a5c4a0-c09712dd-0019fec0.jpg	small bilateral effusions.
MIMIC-CXR-JPG/2.0.0/files/p17131877/s54362658/3a6e3a3e-495cb420-6dd69090-9cd4d215-34f15157.jpg	ap chest compared to : tip of the endotracheal tube is no more than <num> cm above the carina, <num> cm below optimal placement. lungs are grossly clear. heart size is normal. there is no pulmonary edema. pleural effusions are small if any. upper enteric drainage tube ends in the stomach. right subclavian line in the low svc. no pneumothorax. dr was paged at as soon as the findings were recognized.
MIMIC-CXR-JPG/2.0.0/files/p14836998/s54355645/5a4367e5-1732ad3e-f4cac33a-30d62c98-012eb1e4.jpg	mild interstitial pulmonary edema.
MIMIC-CXR-JPG/2.0.0/files/p15561274/s55765261/18e4f494-6397b482-f4015908-4508efdd-3fddd405.jpg	cardiomegaly without pulmonary edema. no signs of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p17074984/s53108643/a32368f8-aa5c0bda-88492b2c-261384c0-7adf0eb5.jpg	in comparison with the earlier study of this date, there has been placement of a left ij catheter that extends to the mid portion of the svc. no evidence of pneumothorax or significant change other than lower lung volumes.
MIMIC-CXR-JPG/2.0.0/files/p12725946/s55646019/d1e55e46-720ab9bc-a1f1883d-7254cd35-65615475.jpg	no retrocardiac consolidation to correlate with the consolidation seen on recent cta chest. this may reflect interval resolution of atelectasis/lower lobe collapse.
MIMIC-CXR-JPG/2.0.0/files/p11825462/s57519039/8aa5a71b-800d656a-4fb98eee-2f19ad58-caaa25eb.jpg	interval placement of a right internal jugular central venous catheter terminating in the low svc without evidence of pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p17457987/s57177782/436b7a5c-0bedd1ba-33c44264-23354807-2911315b.jpg	no acute cardiopulmonary abnormality. no free air is detected under the diaphragms.
MIMIC-CXR-JPG/2.0.0/files/p11070517/s52906797/0939b1a2-5d67daee-fe9e9093-5cdb1647-0b0a6a51.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11213912/s50943351/0e74cd15-68482b9d-288ade5a-03324254-89130b98.jpg	no significant interval change with multifocal, bilateral airspace opacities and bilateral pleural effusions. findings could be related to edema and/or infection.
MIMIC-CXR-JPG/2.0.0/files/p17399675/s51018731/6dc4e318-bf75fd94-13718c0e-de8070b2-8fbcf05e.jpg	pa and lateral chest compared to : atelectasis persists, though improved in both the left base and in the right middle lobe. there is no pulmonary edema or any new lung findings to suggest infection. as noted previously, diagnosis of pulmonary embolus needs to be considered in the setting of the recent history of chest pain, hemoptysis and radiographic findings of atelectasis. mild cardiomegaly is stable. there is no pulmonary vascular congestion or other indirect evidence of cardiac decompensation.
MIMIC-CXR-JPG/2.0.0/files/p19855099/s58589168/65c7d02d-15138714-b8a8310d-b3d68fe5-a3852f32.jpg	ap chest compared to through : right lower lobe consolidation and pulmonary vascular congestion have been present on virtually all prior chest radiographs, including those since. mild pulmonary edema worsened since. severe cardiomegaly may have improved minimally. small pleural effusions are pronounced. lower lobe opacification has worsened. this could be just edema and atelectasis, but radiographically, i cannot exclude pneumonia. left lower lobe is barely visible, obscured by the heart. no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p18867536/s52682055/efa860b8-ca0d2ded-57a58d3b-242b6e42-c4cd96bd.jpg	increased interstitial markings despite limitations of technique which could represent edema although chronic underlying interstitial process or combination both is possible.
MIMIC-CXR-JPG/2.0.0/files/p17187763/s59943519/8304ce5c-ae43fbff-1ce37459-862718d9-b1421abb.jpg	chronic stable right middle lobe and left lower lobe opacities, likely scarring from prior parenchymal process. no radiographic evidence of acute cardiopulmonary process. results were conveyed via telephone to dr by dr on at within <num> minutes of observation of findings.
MIMIC-CXR-JPG/2.0.0/files/p10968669/s52982345/9cf3b5aa-9ff5eee5-09982cd1-eeebf2f2-b0cb2d5a.jpg	since the prior study there has been interval minimal change in enlarged cardiac silhouette, mediastinal contour which is overall unremarkable and vascular enlargement. no focal consolidations to suggest infection are present. there is no pleural effusion or pneumothorax seen. vascular stent is projecting over most likely left subclavian vein
MIMIC-CXR-JPG/2.0.0/files/p13983282/s58321334/c5a0ca6c-bf1cdc01-0198056c-4112f86d-4b8cdd4e.jpg	in comparison with the study of , there is little change in the appearance of heart and lungs. again there is enlargement of the cardiac silhouette with significant pulmonary edema and possible small pleural effusions.
MIMIC-CXR-JPG/2.0.0/files/p16476559/s54299496/ff4a1d07-b8996fcb-8b5830a8-38f8a6cf-803b2760.jpg	the patient is post cabg. mild-to-moderate cardiomegaly is unchanged. there is central pulmonary vascular congestion with mild interstitial edema, more pronounced on the right, new since. there is no pneumothorax or pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p17076416/s50444408/4d1d0d38-833b46f4-d84079f8-f8eac1d3-b61364a7.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17487379/s50835290/293cec0b-a98adf65-18822de8-134f09d9-3d310326.jpg	ap chest compared to : pneumopericardium is no longer present. lung volumes remain quite low, exaggerating caliber of the cardiac silhouette which is probably normal. unexplained is progressive distention of the azygos vein which had dilated between and , even with pericardial drainage catheter in place. left internal jugular line ends low in the svc. small right apical pneumothorax has decreased since.
MIMIC-CXR-JPG/2.0.0/files/p11083931/s52328686/b9139fa7-b9816228-be9bf1c1-71892b0f-d6108dd1.jpg	mild peribronchial thickening may suggest small airways disease. no focal consolidation suggestive of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p14361990/s56808826/7f8c8253-a5022b7b-fde8190f-8e246f5f-10476a06.jpg	no acute findings. calcified pleural plaque likely accounts for left mid lung opacity.
MIMIC-CXR-JPG/2.0.0/files/p19706109/s53062467/8b833106-45f46b15-5ce643d9-7e0c4c45-c9e6ed67.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p15770081/s59577641/c39b8b82-6855be54-b830e409-0775ddf6-06154e4f.jpg	interval development of a tiny left pleural effusion. no consolidation or right pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p13689440/s53623640/cb4b85a1-96f17f95-14f96422-e5ab823a-7d144bd5.jpg	right internal jugular central venous catheter identified terminating within the distal at see in improved position, previously projecting over the right atrium. no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p18839030/s55492152/294ad2f5-d9ddebea-499a974b-ce7a7abd-ebfae322.jpg	new interstitial opacities in the lingula and right lower lung, most likely representing pneumonia. no evidence of pulmonary edema. findings were discussed by dr with dr by phone at on.
MIMIC-CXR-JPG/2.0.0/files/p17790915/s59192241/7dfb4cfc-76447ef1-94172ceb-59dfa23e-df68a33f.jpg	ap chest compared to : feeding tube with the wire stylet in place passes beyond the upper stomach and out of view. a large right pleural effusion may have increased in the interim. heart size top normal. mediastinal veins are dilated, due in part to non-erect positioning, but suggestive of volume overload, since pulmonary vessels are also distended.
MIMIC-CXR-JPG/2.0.0/files/p18338007/s50094334/0d3ff5e0-5202a70f-86af9d84-eec64254-845e87d4.jpg	low lung volumes. no focal opacification concerning for pneumonia. mild prominence of the pulmonary vasculature may indicate mild volume overload.
MIMIC-CXR-JPG/2.0.0/files/p17718978/s55103957/476118d2-6429a0fe-01654e36-5111d5fd-8acb65b5.jpg	stable cardiomegaly and mild worsened pulmonary edema compared to prior of.
MIMIC-CXR-JPG/2.0.0/files/p17700773/s58035143/092bde3c-a100f930-3aaf838e-0abdd990-236595f6.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p16337817/s55861457/9272a705-d2c59865-76e41834-7ff93126-d14298c1.jpg	no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p12293804/s57187009/31a8bafe-08f7ae37-c113888a-1c4e2847-8b9d99ef.jpg	no pulmonary edema. minimal bibasilar linear atelectasis. rest of the findings are unchanged compared to the prior radiograph.
MIMIC-CXR-JPG/2.0.0/files/p14658826/s50965396/972c2f22-0c530ea1-e3dfc6e2-67139638-b50a8ed3.jpg	diffuse reticulonodular interstitial pattern is chronic. due to the the severity of this chronic pathology, detection of subtle pneumonia would be difficult. there is no obvious large pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p12115320/s57693145/10e5db25-e610e768-1208ea2f-e0786de5-2feb9a3f.jpg	new right upper lobe opacity, in the appropriate clinical setting this can represent early consolidation. interval improvement in the left retrocardiac opacity.
MIMIC-CXR-JPG/2.0.0/files/p12468016/s57550787/29c32410-df11d9bc-13b7786c-cc69afb9-fe6ad1e4.jpg	persistent bibasilar opacities, somewhat improved on the right, potentially due to atelectasis.
MIMIC-CXR-JPG/2.0.0/files/p16870968/s55037151/517c7887-8aa3e730-d2efb48e-0ed3586e-c4f65090.jpg	no definite acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14972853/s58002951/44655c1f-6b3ecf77-37ed9e12-3e0c4e61-33c81890.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10758777/s50988857/43d86b98-71a4edac-6b65c173-96db3781-8a0a4458.jpg	resolved chf. cardiomegaly. otherwise, normal chest radiograph.
MIMIC-CXR-JPG/2.0.0/files/p19526163/s58930190/f11554c7-10a5a700-c54a4a94-ae52cde6-2b2bddc1.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p16283494/s53674426/1055167f-38810333-2c1d26f3-51510ab8-c915440e.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p13436774/s51408459/d4c2823d-341fe18f-c905bdcc-e33e78ec-40c358ff.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p19745383/s57245273/df11b94b-12e94ffc-0529404c-b8b06052-1f5cead1.jpg	no focal consolidation or pulmonary nodule.
MIMIC-CXR-JPG/2.0.0/files/p13714231/s50089855/f8d44f0e-026e908a-971c144c-b5b29bd3-7a7d1168.jpg	bibasilar opacities and bronchiectasis has worsened, and is worrisome for developing pneumonia or aspiration.
MIMIC-CXR-JPG/2.0.0/files/p18596679/s54197992/1601366e-693c9ec3-4362353c-3cde27e1-2e22d7a6.jpg	low lung volumes with bibasilar opacities most likely due to atelectasis, though component of aspiration or infection cannot be entirely excluded.
MIMIC-CXR-JPG/2.0.0/files/p11833476/s51690062/a75dc4aa-e3f30908-4f607697-81b78a01-00e9853d.jpg	multifocal infiltrative pulmonary abnormality worsened between and , has worsened slightly since. most severe involvement is in the right upper lobe, suggesting pneumonia. remainder is probably edema. left pleural or extrapleural hematomas unchanged in volume. mild cardiac enlargement stable. no new pleural effusion or pneumothorax. left apical pleural and basal drains are in place. right pic line ends in the mid svc. feeding tube passes into the stomach and out of view.
MIMIC-CXR-JPG/2.0.0/files/p12324852/s52935058/595775e2-deb939b8-631ebb4e-ff16824a-c7e9737c.jpg	no acute findings in the chest.
MIMIC-CXR-JPG/2.0.0/files/p19158091/s59710316/93141176-21f63054-d2edf321-84845732-f94a9d4c.jpg	in comparison with the study of , there again are extensive pleural calcifications consistent with asbestos no evidence of acute pneumonia or vascular congestion.
MIMIC-CXR-JPG/2.0.0/files/p16015242/s54507692/5e2f59ed-2785dd21-2fca1554-715d7cc7-2a11de47.jpg	in comparison with the study of , following right lower lobe lung biopsy there is no evidence of pneumothorax. no evidence of acute pneumonia or vascular congestion.
MIMIC-CXR-JPG/2.0.0/files/p18302636/s52458379/4d51cc03-838e94b4-68f6808a-975a2e59-a8765764.jpg	no acute process
MIMIC-CXR-JPG/2.0.0/files/p17637826/s50660966/a08b1ad3-8175e21d-1bd6af8f-ed2e617d-e5379c79.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11954132/s55463312/8e7696db-d7a055fc-3bca766f-583b0093-61bf41bc.jpg	hyperinflated lungs in keeping with history of emphysema. no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p12743864/s53740085/eafbb7dc-b4b224c9-a4b7c351-6aa504e9-c4812cd1.jpg	no significant interval change with low lung volumes and bilateral pleural effusions and bibasilar atelectasis. extensive skeletal metastases
MIMIC-CXR-JPG/2.0.0/files/p14704955/s57908404/28e89126-7f5fc227-3dee41a3-bb5a234d-5516d248.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p16511261/s53760965/eebbe6c1-6dc1bfdd-7f5591b5-7a9e0795-67f2ba0b.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10594556/s57116398/8ddf91e4-72e1b05a-a8948f81-6aa2ba74-10bab131.jpg	left-sided pneumothorax as seen on prior ct scan. dense left basilar opacity compatible with left lower lobe collapse and opacity in the left upper lobe as well.
MIMIC-CXR-JPG/2.0.0/files/p15836305/s53054315/59d6808d-eae0e854-669e207e-d250d75c-b1072f5a.jpg	in comparison with the study of , there is no definite pneumothorax or other acute cardiopulmonary disease. what appear to be change sutures are seen in the left upper zone.
MIMIC-CXR-JPG/2.0.0/files/p12654952/s51974684/4558fbeb-af8e93c5-17535564-3c31548a-6a4ca9f7.jpg	low lung volumes with bibasilar atelectasis. streaky opacification at the right cardiophrenic angle is most likely vascular crowding due to low lung volumes, however, infection cannot be excluded given the appropriate clinical circumstance.
MIMIC-CXR-JPG/2.0.0/files/p19919951/s55044971/93935300-fd0d3208-b413c4cc-ffa5d420-7c35a8ec.jpg	probable mild cardiomegaly. minimal patchy opacity left base. while this likely represents atelectasis, in the appropriate clinical setting, the differential diagnosis could include an early pneumonic infiltrate.
MIMIC-CXR-JPG/2.0.0/files/p11358361/s59259653/847bbc16-7193df96-d93eeeab-2d74240f-3ab2e28b.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15378749/s55840707/9a2334ea-f1e0766b-60816c58-7733055a-3e423b7a.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15922867/s52626544/93a70551-950c5868-06740df6-f1dcba17-324e6827.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p12618901/s59100053/10dd4935-1516a363-731f5b04-22291d01-4e12a7e3.jpg	a small, curvilinear opacity projects over the right lung apex. this likely represents an external structure, however a repeat radiograph after removal or repositioned of external structures is recommended to exclude a small right apical pneumothorax. diffuse, ground-glass and reticular opacity, predominantly in the left hemithorax, corresponding to ground-glass and reticular opacities from ct. these appear minimally improved in comparison to. left port-a-cath ends at the mid svc.
MIMIC-CXR-JPG/2.0.0/files/p18257430/s57594646/adf80189-e7f52566-80af88db-4cb4dc22-8c2807eb.jpg	right lower lobe pneumonia. recommended followup chest radiograph in weeks after treatment to document resolution.
MIMIC-CXR-JPG/2.0.0/files/p16315929/s54395658/20079d38-771ee1fd-c123f08c-58e71167-4cc8c7f1.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p10124346/s58826769/a20cc217-bcbc624a-2c94ebf7-0478eaff-f07a012f.jpg	low lung volumes which accentuate the bronchovascular markings. patchy opacity projecting over the lateral left lung base on the frontal view, not well substantiated on the lateral view, may be due to atelectasis versus infection.
MIMIC-CXR-JPG/2.0.0/files/p11655271/s54140754/e1cb66c7-63acc795-b727594a-7c37e3df-b649012d.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15711512/s50106086/6b6d29f0-3d9b77a6-9aa260d0-7569d3e8-5faa972e.jpg	in comparison to previous radiograph of <num> days earlier, pulmonary vascular congestion persists, but mild edemahas resolved in the interval. there are no areas of consolidation to suggest the presence of pneumonia, and no pleural effusion or pneumothorax is detected.
MIMIC-CXR-JPG/2.0.0/files/p17374256/s58710716/0cb5a54b-2f80ac5a-49d91afb-39a70a41-705319f6.jpg	increased interstitial edema and small bilateral pleural effusions, greater on the left than the right, suggesting heart failure. possible new left lower lobe pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p12606543/s51793077/2b2a2250-babd272e-1bc26d97-b6b364fa-e61b2365.jpg	moderate pulmonary edema is unchanged. moderate cardiomegaly is stable.
MIMIC-CXR-JPG/2.0.0/files/p19221748/s56292741/2c50314e-052a4fea-da2139a5-4af7aec7-3cb4cd44.jpg	ap chest compared to. right hemidiaphragm is chronically elevated and permits upward migration of the hepatic flexure of the colon, which is still in the abdomen. pulmonary vascular engorgement and mild pulmonary edema have developed since. progressive elevation of the left lung base is largely due to the elevation of the left hemidiaphragm. there may also be a new small left subpulmonic pleural effusion. two right pleural drains are still in place and there is minimal if any right pleural effusion and no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p15349002/s50635527/7aba7c4b-b493ea1b-4d2947e7-991087a8-1868dff3.jpg	in comparison with the study of , the current study is extremely limited due to low lung volumes and the chin and other devices about the patient greatly obscuring detail. there is asymmetric opacification at the left base with poor definition of the hemidiaphragm. this could reflect merely atelectasis and effusion postoperatively. however, the possibility of superimposed aspiration would have to be seriously considered in the appropriate clinical setting.
MIMIC-CXR-JPG/2.0.0/files/p15126244/s56905780/d88de143-c8ed756d-4b3d54c0-8e151e98-cb344515.jpg	no pneumonia, edema or effusion.
MIMIC-CXR-JPG/2.0.0/files/p19046950/s55956132/76e73a13-f2cae15d-e83711b4-1280767d-85e31015.jpg	ng tube tip isout of view, below the diaphragm. right picc tip is in the cavoatrial junction. cardiac size is normal. the aorta is tortuous. there are low lung volumes. multifocal atelectasis have improved in the right upper lobe, stable in the lower lobes bilaterally left greater than right. there is no pneumothorax or large effusions
MIMIC-CXR-JPG/2.0.0/files/p19291544/s50048825/39044a13-56699970-37d70eb4-107ad9d6-238a3889.jpg	nasogastric tube ends in the stomach.
MIMIC-CXR-JPG/2.0.0/files/p17876390/s50567998/cf01a48c-6314f3a1-b010a8d5-fbb2f73a-17b172bb.jpg	no infiltrates
MIMIC-CXR-JPG/2.0.0/files/p16228877/s58622466/aaa5addf-f7d4dc1f-e222978d-5b65c8ff-946c6e71.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p19157078/s54728144/c890e703-3db9b792-4c6affe3-c5298611-801761f6.jpg	huge midline gastrointestinal hernia, probably through the esophageal hiatus, is chronic, responsible for considerable atelectasis at the left base. considerable right lower lobe atelectasis is also long-standing. the upper lungs are clear. heart size is indeterminate but probably not enlarged. no pneumothorax. although no acute fracture or other chest wall lesion is seen, conventional chest radiographs are not sufficient for detection or characterization of such abnormalities. if the demonstration of such a chest cage abnormality is clinically warranted, the location of any referrable focal findings should be clearly marked and imaged with either bone detail views or ct scanning.
MIMIC-CXR-JPG/2.0.0/files/p16979251/s58226911/e64a8c04-fe5c1cc4-c1feb3e2-b213e452-38327706.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p17425397/s52335411/995e27e5-cd637ec6-abe65709-a3c05ceb-ac6294c3.jpg	no evidence of acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17126702/s57826861/13f2f15e-7b4782a4-acf251f7-78dc8e4c-c31977fb.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17122884/s54258365/b0fe2766-4442aa04-635acbaf-ccced98e-da041653.jpg	multifocal pneumonia. no prior studies are available for comparison.
MIMIC-CXR-JPG/2.0.0/files/p11585967/s53785490/887a8d81-39b3820e-07d763f9-f7c71abc-e41a6488.jpg	pacemaker with single lead in the right ventricle, without pneumothorax or evidence of other complication. possible increase in severe cardiomegaly, but pulmonary vascular engorgement has improved and there is no edema.
MIMIC-CXR-JPG/2.0.0/files/p17025318/s51740951/1e87f62e-1763b20e-6a9d7aef-a394f78e-0741698a.jpg	ap single view of the chest shows interval reduction of left apical pneumothorax. left lung base nodular and patchy opacities have increased and compatible with localized contusion or atelectasis. the left pleural drain has oblique orientation, and it might be intrafissural. small left posterior pleural effusion seems comparable to that described in ct of. right lung is clear. cardiomediastinal silhouette is normal.
MIMIC-CXR-JPG/2.0.0/files/p15808118/s57730086/ee1758ae-af98a497-9a3e707f-50347390-beaa0ebc.jpg	no radiographic evidence for acute cardiopulmonary process.

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no evidence of acute cardiopulmonary process.

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no acute cardiopulmonary process.

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no acute intrathoracic process.

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the left-sided chest tube has been removed. no pneumothoraces are seen. there is volume loss and loculated pleural fluid along the left lateral chest, unchanged. right lung appears well aerated.

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no evidence of interstitial prominence. no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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left basilar atelectasis.

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bilateral lower lobe consolidation is unchanged since earlier in the day consistent with persistent pneumonia. upper lungs are clear. there is no pulmonary edema. pleural effusions a small if any. heart size top-normal unchanged. et tube in standard placement. nasogastric drainage tube passes into the stomach and out of view. left pic line ends in the low svc. right jugular line ends in the upper svc. ventriculoperitoneal shunt traverses the right neck chest and upper abdomen common passing out of view. incidental note is made of a large granulomatous lymph node calcifications in the mediastinum and left hilus and a smaller calcification in the left lung.

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no radiographic evidence of pneumonia.

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normal. no parenchymal abnormality.

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in comparison with the study of , the endotracheal tube and nasogastric tube have been removed. the right ij catheter is unchanged. there is again a large right pleural effusion with compressive basilar atelectasis as well as volume loss in the left lower lobe with smaller effusion. the left perihilar opacity is unchanged.

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overall, appearances are unchanged compared to the prior study. persistent pneumoperitoneum and loculated left basal pneumothorax.

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new retrocardiac opacity on the lateral view potentially due to atelectasis however it was not seen on prior film with similar inspiratory effort, consolidation would also be possible. please correlate clinically.

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no radiographic evidence for acute cardiopulmonary process.

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the heart remains enlarged. mediastinal contours are unremarkable. the aorta is unfolded and tortuous. lungs are well inflated without evidence of focal airspace consolidation to suggest pneumonia. no pleural effusions or pneumothorax. no evidence of pulmonary edema. mild eventration of the right hemidiaphragm.

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there continues to be a layering left effusion with associated airspace opacity which likely reflects atelectasis. there is likely a smaller right effusion with linear and patchy opacities at the base also suggestive of atelectasis. possible minimal to mild interstitial edema. overall cardiac and mediastinal contours are likely stable although difficult to assess due to the patient rotation on the current study. no pneumothorax.

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as compared to radiograph, widespread pulmonary opacities have apparently progressed and likely reflect worsening acute lung disease process such as infectious pneumonia with possible associated ards superimposed upon chronic interstitial lung disease evident on prior outside ct of. no other relevant changes since recent study.

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small bilateral effusions.

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ap chest compared to : tip of the endotracheal tube is no more than <num> cm above the carina, <num> cm below optimal placement. lungs are grossly clear. heart size is normal. there is no pulmonary edema. pleural effusions are small if any. upper enteric drainage tube ends in the stomach. right subclavian line in the low svc. no pneumothorax. dr was paged at as soon as the findings were recognized.

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mild interstitial pulmonary edema.

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cardiomegaly without pulmonary edema. no signs of pneumonia.

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in comparison with the earlier study of this date, there has been placement of a left ij catheter that extends to the mid portion of the svc. no evidence of pneumothorax or significant change other than lower lung volumes.

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no retrocardiac consolidation to correlate with the consolidation seen on recent cta chest. this may reflect interval resolution of atelectasis/lower lobe collapse.

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interval placement of a right internal jugular central venous catheter terminating in the low svc without evidence of pneumothorax.

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no acute cardiopulmonary abnormality. no free air is detected under the diaphragms.

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no acute cardiopulmonary process.

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no significant interval change with multifocal, bilateral airspace opacities and bilateral pleural effusions. findings could be related to edema and/or infection.

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pa and lateral chest compared to : atelectasis persists, though improved in both the left base and in the right middle lobe. there is no pulmonary edema or any new lung findings to suggest infection. as noted previously, diagnosis of pulmonary embolus needs to be considered in the setting of the recent history of chest pain, hemoptysis and radiographic findings of atelectasis. mild cardiomegaly is stable. there is no pulmonary vascular congestion or other indirect evidence of cardiac decompensation.

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ap chest compared to through : right lower lobe consolidation and pulmonary vascular congestion have been present on virtually all prior chest radiographs, including those since. mild pulmonary edema worsened since. severe cardiomegaly may have improved minimally. small pleural effusions are pronounced. lower lobe opacification has worsened. this could be just edema and atelectasis, but radiographically, i cannot exclude pneumonia. left lower lobe is barely visible, obscured by the heart. no pneumothorax.

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increased interstitial markings despite limitations of technique which could represent edema although chronic underlying interstitial process or combination both is possible.

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chronic stable right middle lobe and left lower lobe opacities, likely scarring from prior parenchymal process. no radiographic evidence of acute cardiopulmonary process. results were conveyed via telephone to dr by dr on at within <num> minutes of observation of findings.

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since the prior study there has been interval minimal change in enlarged cardiac silhouette, mediastinal contour which is overall unremarkable and vascular enlargement. no focal consolidations to suggest infection are present. there is no pleural effusion or pneumothorax seen. vascular stent is projecting over most likely left subclavian vein

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in comparison with the study of , there is little change in the appearance of heart and lungs. again there is enlargement of the cardiac silhouette with significant pulmonary edema and possible small pleural effusions.

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the patient is post cabg. mild-to-moderate cardiomegaly is unchanged. there is central pulmonary vascular congestion with mild interstitial edema, more pronounced on the right, new since. there is no pneumothorax or pleural effusion.

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no acute cardiopulmonary process.

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ap chest compared to : pneumopericardium is no longer present. lung volumes remain quite low, exaggerating caliber of the cardiac silhouette which is probably normal. unexplained is progressive distention of the azygos vein which had dilated between and , even with pericardial drainage catheter in place. left internal jugular line ends low in the svc. small right apical pneumothorax has decreased since.

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mild peribronchial thickening may suggest small airways disease. no focal consolidation suggestive of pneumonia.

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no acute findings. calcified pleural plaque likely accounts for left mid lung opacity.

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no acute intrathoracic process.

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interval development of a tiny left pleural effusion. no consolidation or right pleural effusion.

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right internal jugular central venous catheter identified terminating within the distal at see in improved position, previously projecting over the right atrium. no pneumothorax.

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new interstitial opacities in the lingula and right lower lung, most likely representing pneumonia. no evidence of pulmonary edema. findings were discussed by dr with dr by phone at on.

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ap chest compared to : feeding tube with the wire stylet in place passes beyond the upper stomach and out of view. a large right pleural effusion may have increased in the interim. heart size top normal. mediastinal veins are dilated, due in part to non-erect positioning, but suggestive of volume overload, since pulmonary vessels are also distended.

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low lung volumes. no focal opacification concerning for pneumonia. mild prominence of the pulmonary vasculature may indicate mild volume overload.

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stable cardiomegaly and mild worsened pulmonary edema compared to prior of.

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no acute intrathoracic process.

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no pneumothorax.

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no pulmonary edema. minimal bibasilar linear atelectasis. rest of the findings are unchanged compared to the prior radiograph.

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diffuse reticulonodular interstitial pattern is chronic. due to the the severity of this chronic pathology, detection of subtle pneumonia would be difficult. there is no obvious large pneumonia.

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new right upper lobe opacity, in the appropriate clinical setting this can represent early consolidation. interval improvement in the left retrocardiac opacity.

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persistent bibasilar opacities, somewhat improved on the right, potentially due to atelectasis.

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no definite acute cardiopulmonary process.

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no acute cardiopulmonary process.

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resolved chf. cardiomegaly. otherwise, normal chest radiograph.

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no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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no focal consolidation or pulmonary nodule.

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bibasilar opacities and bronchiectasis has worsened, and is worrisome for developing pneumonia or aspiration.

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low lung volumes with bibasilar opacities most likely due to atelectasis, though component of aspiration or infection cannot be entirely excluded.

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multifocal infiltrative pulmonary abnormality worsened between and , has worsened slightly since. most severe involvement is in the right upper lobe, suggesting pneumonia. remainder is probably edema. left pleural or extrapleural hematomas unchanged in volume. mild cardiac enlargement stable. no new pleural effusion or pneumothorax. left apical pleural and basal drains are in place. right pic line ends in the mid svc. feeding tube passes into the stomach and out of view.

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no acute findings in the chest.

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in comparison with the study of , there again are extensive pleural calcifications consistent with asbestos no evidence of acute pneumonia or vascular congestion.

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in comparison with the study of , following right lower lobe lung biopsy there is no evidence of pneumothorax. no evidence of acute pneumonia or vascular congestion.

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no acute process

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no acute cardiopulmonary process.

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hyperinflated lungs in keeping with history of emphysema. no evidence of pneumonia.

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no significant interval change with low lung volumes and bilateral pleural effusions and bibasilar atelectasis. extensive skeletal metastases

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no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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left-sided pneumothorax as seen on prior ct scan. dense left basilar opacity compatible with left lower lobe collapse and opacity in the left upper lobe as well.

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in comparison with the study of , there is no definite pneumothorax or other acute cardiopulmonary disease. what appear to be change sutures are seen in the left upper zone.

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low lung volumes with bibasilar atelectasis. streaky opacification at the right cardiophrenic angle is most likely vascular crowding due to low lung volumes, however, infection cannot be excluded given the appropriate clinical circumstance.

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probable mild cardiomegaly. minimal patchy opacity left base. while this likely represents atelectasis, in the appropriate clinical setting, the differential diagnosis could include an early pneumonic infiltrate.

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no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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no acute cardiopulmonary process.

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a small, curvilinear opacity projects over the right lung apex. this likely represents an external structure, however a repeat radiograph after removal or repositioned of external structures is recommended to exclude a small right apical pneumothorax. diffuse, ground-glass and reticular opacity, predominantly in the left hemithorax, corresponding to ground-glass and reticular opacities from ct. these appear minimally improved in comparison to. left port-a-cath ends at the mid svc.

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right lower lobe pneumonia. recommended followup chest radiograph in weeks after treatment to document resolution.

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no acute intrathoracic process.

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low lung volumes which accentuate the bronchovascular markings. patchy opacity projecting over the lateral left lung base on the frontal view, not well substantiated on the lateral view, may be due to atelectasis versus infection.

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no acute cardiopulmonary process.

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in comparison to previous radiograph of <num> days earlier, pulmonary vascular congestion persists, but mild edemahas resolved in the interval. there are no areas of consolidation to suggest the presence of pneumonia, and no pleural effusion or pneumothorax is detected.

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increased interstitial edema and small bilateral pleural effusions, greater on the left than the right, suggesting heart failure. possible new left lower lobe pneumonia.

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moderate pulmonary edema is unchanged. moderate cardiomegaly is stable.

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ap chest compared to. right hemidiaphragm is chronically elevated and permits upward migration of the hepatic flexure of the colon, which is still in the abdomen. pulmonary vascular engorgement and mild pulmonary edema have developed since. progressive elevation of the left lung base is largely due to the elevation of the left hemidiaphragm. there may also be a new small left subpulmonic pleural effusion. two right pleural drains are still in place and there is minimal if any right pleural effusion and no pneumothorax.

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in comparison with the study of , the current study is extremely limited due to low lung volumes and the chin and other devices about the patient greatly obscuring detail. there is asymmetric opacification at the left base with poor definition of the hemidiaphragm. this could reflect merely atelectasis and effusion postoperatively. however, the possibility of superimposed aspiration would have to be seriously considered in the appropriate clinical setting.

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no pneumonia, edema or effusion.

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ng tube tip isout of view, below the diaphragm. right picc tip is in the cavoatrial junction. cardiac size is normal. the aorta is tortuous. there are low lung volumes. multifocal atelectasis have improved in the right upper lobe, stable in the lower lobes bilaterally left greater than right. there is no pneumothorax or large effusions

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nasogastric tube ends in the stomach.

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no infiltrates

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no acute cardiopulmonary process.

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huge midline gastrointestinal hernia, probably through the esophageal hiatus, is chronic, responsible for considerable atelectasis at the left base. considerable right lower lobe atelectasis is also long-standing. the upper lungs are clear. heart size is indeterminate but probably not enlarged. no pneumothorax. although no acute fracture or other chest wall lesion is seen, conventional chest radiographs are not sufficient for detection or characterization of such abnormalities. if the demonstration of such a chest cage abnormality is clinically warranted, the location of any referrable focal findings should be clearly marked and imaged with either bone detail views or ct scanning.

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no acute intrathoracic process.

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no evidence of acute cardiopulmonary process.

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no acute cardiopulmonary process.

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multifocal pneumonia. no prior studies are available for comparison.

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pacemaker with single lead in the right ventricle, without pneumothorax or evidence of other complication. possible increase in severe cardiomegaly, but pulmonary vascular engorgement has improved and there is no edema.

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ap single view of the chest shows interval reduction of left apical pneumothorax. left lung base nodular and patchy opacities have increased and compatible with localized contusion or atelectasis. the left pleural drain has oblique orientation, and it might be intrafissural. small left posterior pleural effusion seems comparable to that described in ct of. right lung is clear. cardiomediastinal silhouette is normal.

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no radiographic evidence for acute cardiopulmonary process.