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MIMIC-CXR-JPG/2.0.0/files/p13760104/s53420260/e49b5fab-d09d99be-155abd07-9fd1ae1d-e4984599.jpg
left lower lobe pneumonia. dr was informed of these findings at <num> pm on by dr phone.
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hyperinflated lungs without superimposed pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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no acute findings in the chest.
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left-sided ij terminates in the mid svc. interval worsening of pneumonia.
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the tip of the tunneled catheter is in the mid to lower portion the svc. there is no evidence of acute pneumonia or vascular congestion or appreciable change from the study of.
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no acute intrathoracic process, specifically no signs of pneumonia.
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mild alveolar pulmonary edema and cardiomegaly, new from.
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no acute cardiopulmonary process.
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no focal opacity concerning for pneumonia.
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in comparison with the study of , there has been a slight change in the appearance of the chest to, which has its tube pointing upwards. opacification at the right base is unchanged and there is no evidence of pneumothorax. remainder of the study is little different from the previous
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the nasogastric tube remains in place but only can be seen to the level the mid esophagus and therefore the positioning cannot be assessed on this image. a right internal jugular central line continues to have its tip in the proximal right atrium. there has been slight interval improvement in lung volumes with residual patchy opacities in both upper lobes and in the left retrocardiac region. these findings would be concerning for pneumonia, less likely pulmonary edema given the distribution. clinical correlation is advised. there is slight improved aeration at the right lung base. overall cardiac and mediastinal contours are stable. no pneumothorax.
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findings suggestive of right middle lobe atelectasis, component of infection is possible. no definite rib fracture although if desired dedicated rib series could be obtained.
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on the lateral view, there is subtle retrosternal opacity which may be due to atelectasis although consolidation due to infection is not excluded in the appropriate clinical setting. no focal consolidation is seen elsewhere. attention at follow-up.
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no acute cardiopulmonary process.
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no focal consolidation concerning for pneumonia. mild pulmonary vascular prominence appears similar since.
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obscuration of the right hemidiaphragm and right lower lobe opacity are likely a combination of atelectasis and pleural fluid. superimposed infection is not excluded.
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no evidence of pneumonia. resolved right lung opacities.
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chronic interstitial abnormality within the lung bases which could reflect chronic aspiration given the presence of punctate radiopaque material in the right lung base possibly representing aspirated barium. findings could be better characterized with a dedicated chest ct on a nonemergent basis.
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new right infrahilar opacity, suspicious for pneumonia. recommendation(s): follow-up chest radiograph in <num> weeks to document resolution.
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<num>) background copd. no acute pulmonary process identified. <num>) no displaced rib fracture or obvious lytic or sclerotic lesion detected. <num>) evidence of prior surgery.
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in comparison with the study of , the tracheostomy tube left picc line, and dual-channel pacer are unchanged. the increased opacification at the right base has improved, with little change in the retrocardiac opacification. although these findings most likely reflect pleural effusion and compressive atelectasis, in the appropriate clinical setting it would be difficult to exclude superimposed pneumonia.
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no acute cardiopulmonary process.
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support and monitoring devices are unchanged in position, and cardiomediastinal contours are stable. slight improvement an asymmetrically distributed heterogeneous lung opacities, affecting the left lung to a greater degree than the right. persistent pneumoperitoneum, likely related to recent peg placement.
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extensive bilateral pleural and pulmonary opacities concerning for metastatic disease with areas of fibrosis. please correlate clinically, ct recommended in the absence of prior imaging to further assess.
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no acute cardiopulmonary process.
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in comparison with the earlier study of this date, there again are low lung volumes with continued widening of the superior mediastinum and bibasilar atelectatic changes. cardiac silhouette is probably mildly enlarged but there is no definite vascular congestion.
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no acute cardiopulmonary process.
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no focal pneumonia.
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normal.
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no acute findings, specifically no evidence of pneumonia.
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no significant change to the appearance of the lungs.
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no acute cardiopulmonary process.
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in comparison with the study of , the monitoring and support devices are essentially unchanged. the diffuse areas of increased opacification bilaterally appear to be slightly decreased. the appearance is consistent with the clinical diagnosis of widespread pneumonia, possibly combined with elevation of pulmonary venous pressure. there also are bilateral layering pleural effusions with underlying compressive basilar atelectasis.
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central venous line tip is at the level of lower svc. heart size and mediastinum are stable. lungs are clear. there is no pleural effusion. there is no pneumothorax.
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right middle lobe pneumonia that has newly occurred as compared to. at the time of observation and dictation, , on , the referring physician,. was paged for notification. the findings were discussed over the telephone.
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ap chest compared to and : previous mild-to-moderate pulmonary edema has substantially cleared. the residual opacification in the left lung base is a candidate for possible pneumonia, and should be followed. pleural effusions are small and unchanged. moderate cardiomegaly is stable. mediastinal vascular engorgement has improved slightly. no pneumothorax.
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no acute cardiopulmonary process.
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persistent but mildly improved moderate pulmonary edema.
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in comparison with the study of , there has been development of a substantial right pleural effusion with underlying compressive atelectasis. significant hyperexpansion of the lungs with underlying emphysema is again noted. the mediastinal mass is progressively smaller.
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right basilar opacity, most consistent with a new pneumonia.
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<num>) interval placement of dobbhoff tube, with radiopaque tip overlying the stomach. this does not pass beyond the pylorus. <num>) bilateral pulmonary opacities, left lower lobe collapse and/or consolidation and small left effusion. the lower right chest wall and right costophrenic angle are excluded from the film. <num>) cardiomegaly, similar to prior. <num>) possible rib fracture lower left chest. clinical correlation requested.
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subtly increased lung markings in the left lower lobe likely reflect atelectasis or scarring and are unchanged from the prior exam in. low lung volumes. no definite pneumonia.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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top normal heart size, otherwise normal.
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possible early right middle lobe pneumonia.
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calcified pleural plaques suggesting prior asbestos exposure. right shoulder dislocation better seen on concurrent shoulder films. no displaced rib fractures seen based on this nondedicated examination.
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mild interstitial pulmonary edema. no focal consolidation.
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in comparison with study of , the patient has taken slightly better inspiration no evidence of vascular congestion, pleural effusion, or acute focal pneumonia
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stable appearance of the chest with no pulmonary edema.
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no radiographic evidence of pneumonia.
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no evidence of pneumonia.
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heart size is normal. mediastinum is normal. lungs are clear. there is no pleural effusion or pneumothorax. overall normal chest radiograph.
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right mid lung opacity may represent early infection. recommend repeat radiograph after treatment to document resolution. left lower lung nodule is probably callus formation at a healing rib fracture. recommend shallow obliques for confirmation. no evidence of congestive heart failure. discussed with dr at am.
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right infrahilar patchy opacity may relate to overlying vascular structures, although small underlying consolidation is not entirely excluded. dedicated pa and lateral views would be helpful for further evaluation if/when patient is able.
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small bilateral pleural effusions are seen on lateral view only. no pneumothorax.
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as compared to the previous radiograph, no relevant change is seen. the left pectoral pacemaker is constant. the lung volumes are low but there is no evidence of pulmonary edema or pneumonia. no pleural effusions.
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no acute cardiopulmonary process.
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no evidence of acute disease.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no comparison. the lung volumes are normal. normal size of the cardiac silhouette. normal hilar and mediastinal contours. no pneumonia, no pulmonary edema, no pleural effusions.
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as compared to the previous image, the known left basal pneumonia has almost completely cleared. only on the lateral radiograph, an area of minimal scarring at the left lower lobe bases is visualized. no pleural effusion. no new focal parenchymal opacities. normal size of the cardiac silhouette.
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low lung volumes makes assessment for hilar lymphadenopathy and evaluation of an apparent left posterior basilar opacity difficult. when the patient's condition permits, pa and lateral radiographs with a deeper inspiration are recommended to better assess the hilar structures and to re-evalute the posterior basal left lower lobe.
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no acute intrathoracic process.
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bilateral pleural effusions, left greater than right. moderate-to-severe cardiomegaly. peripheral parenchymal or pleural opacities bilaterally. these findings appear to be new at least since when the lung bases were visualized on the ct. further evaluation with chest ct is recommended. bilateral widening of the glenohumeral joint spaces may be indicative of rotator cuff laxity. correlation with history and physical examination is recommended.
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compared to chest radiographs :<num>. tip of the endotracheal tube above the upper margin of the clavicles is no less than <num> cm from the carina with the chin elevated. it should not be withdrawn any further. nasogastric drainage tube ends in the mid stomach. lungs are low in volume but clear. normal cardiomediastinal and hilar silhouettes. no pleural abnormality.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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increased size of large left pleural effusions from is expected status post left pneumonectomy. decreased size of small right pleural effusion with resolution of right lower lobe opacity from. resolved subcutaneous emphysema.
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no acute cardiopulmonary process.
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no aspiration pneumonia is seen.
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low lung volumes. no evidence of acute cardiopulmonary process.
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no definite acute cardiopulmonary process.
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right costophrenic angle not fully included on the image. otherwise, no acute cardiopulmonary process. interval re-expansion of the right upper lobe.
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mild bibasilar atelectasis in the setting of low lung volumes. no definite evidence for pneumonia.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process. mild bibasilar atelectasis.
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stable cardiomediastinal silhouette with mild cardiomegaly. no pulmonary edema. emphysema.
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coronary artery calcifications. no evidence of acute cardiopulmonary disease.
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as compared to the previous radiograph, the extent of the left pleural effusion is constant. the effusion is better appreciated on the lateral than on the frontal radiograph. a pre-existing minimal right pleural effusion has almost completely resolved. the areas of peripheral apical e predominant scarring in the lung parenchyma are constant. constant appearance of a retrocardiac atelectasis. borderline size of the cardiac silhouette is unchanged.
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moderate cardiac enlargement, increased compared to the previous study. diffuse bronchiectasis, most severe in the lung bases, but not substantially changed in the interval.
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mild pulmonary edema, not substantially changed in the interval. continued interval improvement in previously seen peripheral right basilar opacity.
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new basilar opacity projecting over the spine likely on the left level may be due to combination of effusion, consolidation or atelectasis. no other change.
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no acute cardiopulmonary process.
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pa and lateral chest reviewed in the absence of chest imaging more recent than a chest ct on : heart is mildly enlarged, but pulmonary vasculature is normal and there is no pleural effusion or evidence of central adenopathy. i see no lung mass or direct evidence of adenopathy or consolidation, but the right hilus is displaced medially and inferiorly. if clinical findings persist, a ct scan would be helpful in excluding a subtle right hilar mass. it will also confirm whether the small hiatus hernia is present, lending additional radiodensity posterior to the heart on the lateral view.
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no acute cardiopulmonary process.
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ap chest at compared to previously symmetric largely interstitial severe infiltrative pulmonary abnormality on accompanied by small bilateral pleural effusions was probably pulmonary edema. today edema has improved somewhat but there are now large areas of consolidation in the right upper lobe anterior segment, left juxta hilar midline and right lung base. these findings suggest widespread pneumonia. pleural effusions are smaller today. heart is not enlarged. et tube is in standard placement. right pic line ends below the svc and an upper enteric drainage tube ends in the mid portion of moderately distended stomach. there is no pneumothorax.
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stable moderate bilateral pleural effusions and right infrahilar pneumonia.
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no acute cardiopulmonary process such as pneumonia. top normal heart size.
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no evidence of pneumonia or other acute cardiopulmonary process.
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persistent right lung base findings consistent with right middle lobe and lower lobe collapse. additional thickening of lateral pleural space on right side, indicative of additional pleural effusion. cause of the collapse is not clear on the plain chest examinations. unless airway exploration is performed to exclude central airway obstruction airway, a chest ct is recommended to diagnose the cause of the major lung collapse.
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over the last <num> hours, moderate-to-severe pulmonary edema has improved.
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no focal lung consolidation. stable appearance of left lower lung pleural thickening and scarring. no pulmonary vascular congestion or edema.
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no acute cardiac or pulmonary process.
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mild pulmonary vascular congestion.