ChayanM/MIMIC-Impression-Dataset · Datasets at Hugging Face

File_Path stringlengths 94 94	Impression stringlengths 1 1.56k
MIMIC-CXR-JPG/2.0.0/files/p13291805/s54365682/84595c4e-b3f7e4d7-18008fac-553d0b26-a5a05555.jpg	left upper extremity picc ends in the low svc.
MIMIC-CXR-JPG/2.0.0/files/p19958337/s55681106/7f7080e6-034fa8cd-dbd354db-52185c6d-62d0bf1e.jpg	ap chest compared to , endotracheal tube, in standard placement, since the chin is flexed. right subclavian line has been repositioned, ends in the right atrium, and would need to be withdrawn approximately <num> cm to reposition it low in the svc, if desired. borderline cardiomegaly and mediastinal vascular engorgement are exaggerated by exceedingly low lung volumes and non-erect positioning. lungs are grossly clear. there is no pneumothorax or appreciable pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p17115490/s56294875/3a78c1b6-99284c67-dd91c3d7-c3b54432-a22a8eae.jpg	interval worsening of near-complete opacification of the left hemithorax, likely related to large left-sided pleural effusion as seen on outside chest ct. new small right pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p17491555/s52457951/07451bfa-9e498d95-8fdc8654-1bd9b869-71b88ce8.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p11206127/s58353141/ecb6fe94-925af862-9ee155df-5ff254dc-cd557df3.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11933456/s58702623/b38ef7fc-638f6ce3-15619b39-46ca0b51-7fc2d826.jpg	increased interstitial markings, most significant at the left lung base, compatible with fibrotic changes likely secondary to underlying interstitial lung disease. pulmonary edema improved overnight. bilateral hilar prominence, which may be secondary to lymphadenopathy or pulmonary arterial enlargement and hypertension.
MIMIC-CXR-JPG/2.0.0/files/p18847956/s50615938/04cf2d55-7bab66e1-c6c25f44-9b3c6d97-8c895044.jpg	no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p10872143/s53328638/dc9a261f-1de2bb99-1ba16c31-b33f1a4f-6bcf7f52.jpg	unchanged moderate right pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p18350594/s53865828/c3ea47d4-a25ad517-0bc0639f-f27ed029-ec552584.jpg	increased interstitial prominence, particularly in the right lung, since the prior study. airway inflammation or atypical infection could be considered in the appropriate clinical setting, although the appearance is not specific. no focal consolidation.
MIMIC-CXR-JPG/2.0.0/files/p18351705/s58138319/e9396e5e-fa89a2bf-209bd8b2-57d91384-3cc591f4.jpg	collapse of the right middle lobe and right lower lobes is new. cardiomegaly cannot be evaluated. there is no evident pneumothorax. the left lung is grossly clear, aside from minimal retrocardiac atelectasis. right chest wall subcutaneous emphysema has improved
MIMIC-CXR-JPG/2.0.0/files/p11845452/s54767309/4c3c76d0-ffa18ec9-4ce248fe-5fe87900-851c9b87.jpg	no evidence of acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17631528/s52848056/4f9820dd-9cf5135d-4a320c4c-f4c3f906-3b59dbf7.jpg	a comparison study of , the patient has taken a better inspiration. no evidence of pneumonia, vascular congestion, or pleural effusion. the configuration of the left port-a-cath is essentially unchanged.
MIMIC-CXR-JPG/2.0.0/files/p12544547/s53725174/52a9615b-5aa6e8e8-2d026154-6eadc929-881d86b1.jpg	bilateral asymmetric pleural thickening with calcifications, suggestive of previous asbestos exposure. in this context, worsening basilar reticular opacities may reflect chronic interstitial lung disease with possible component of superimposed acute edema. follow-up chest radiograph could be performed after diuresis to further clarify this issue. high-resolution ct would also be helpful on a nonemergent basis to better characterize the patient's lung findings.
MIMIC-CXR-JPG/2.0.0/files/p13644746/s58229901/69471570-436c67a3-f9b69d4c-e0dbefde-5d9d34a4.jpg	vague opacity in the right mid-to-lower lung concerning for pneumonia. followup to resolution.
MIMIC-CXR-JPG/2.0.0/files/p12796151/s59815496/2e2f24b5-ac2b448b-7c47b02a-e1847697-b90e60a2.jpg	no acute cardiopulmonary process. no evidence of free air beneath the diaphragms.
MIMIC-CXR-JPG/2.0.0/files/p12304672/s54716937/c0b1f507-65133f6b-02954984-3a2ac0fd-5e0e002c.jpg	probable left fibrothorax with chronic left pleural thickening, loculated fluid, and volume loss. small right pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p15255120/s52100101/078dbfc9-68b7328d-bd812163-43209c07-17d0a8c1.jpg	new right internal jugular central venous line nc in the right atrium at a level approximately <num> cm beyond the estimated location of the superior cavoatrial junction. no pneumothorax pleural effusion or mediastinal widening. mild cardiomegaly is exaggerated by low lung volumes. aside from calcified right upper lobe granuloma, the lungs are essentially clear. no pleural effusion or pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p10374990/s54060668/d97e3634-545c7177-6f6ac486-88ea5566-e82506e6.jpg	compared to prior chest radiographs through. moderate to large bilateral pleural effusions and are larger and mild pulmonary edema has worsened. heart is normal size. t avr is grossly unchanged. no pneumothorax. atelectasis in the right upper lobe developed since and has not improved
MIMIC-CXR-JPG/2.0.0/files/p15958024/s51334198/cba0249b-aaaa471f-ed041338-9198ade0-6ca45d71.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p19889178/s52714326/b75d9e83-f72286de-aba33c59-0b698d3d-65c24319.jpg	interval worsening of cardiopulmonary findings
MIMIC-CXR-JPG/2.0.0/files/p14489759/s53549288/611fa21c-f15b356e-5f21c393-65927356-e5192942.jpg	hyperinflated lungs with evidence of biapical scarring, which most likely relate to copd. large hiatal hernia. no focal consolidation.
MIMIC-CXR-JPG/2.0.0/files/p10542901/s51909348/f72195ed-13193d64-798f8777-ef24850b-8c96b058.jpg	et tube tip is approximately <num> cm above the carina.
MIMIC-CXR-JPG/2.0.0/files/p15706176/s55218108/41366e00-125bef07-40bb1cbe-b061a9e5-a7999ebf.jpg	no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p12589336/s58918792/923aead6-c0e30894-cf88f127-f4b9718b-2b515798.jpg	in comparison with the study of , there again is extensive opacification in the right hilar region upper lung, consistent with the known malignant lesion and collapse of the right upper lobe. the primary tumor and mediastinal lymph nodes are much better appreciated on the previous ct study. there has been re-expansion of the remainder of the right lung with atelectatic streaks at the right base and. moderate subpulmonic pleural effusion is again seen. the left lung is essentially clear.
MIMIC-CXR-JPG/2.0.0/files/p16669225/s52185791/14740c13-6222ffd9-c9bf5b0a-b3ccd8ac-051ca7dc.jpg	no significant interval change.
MIMIC-CXR-JPG/2.0.0/files/p12975896/s59489916/1dc26949-7ba51841-67dc1fc9-562aaad3-659fbc76.jpg	no acute cardiopulmonary pathology.
MIMIC-CXR-JPG/2.0.0/files/p14399272/s53150045/c494152c-79d0a0b9-abc1d9b8-c05cf519-91232209.jpg	increased opacification of the left hemithorax suggesting primarily increase in pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p15757588/s57944782/a2498c2a-ac991b79-5c2568ab-e7d86d50-ef11b157.jpg	small left pleural effusion. retrocardiac opacity may represent compressive atelectasis or aspiration, however pneumonia could be considered in the appropriate clinical setting.
MIMIC-CXR-JPG/2.0.0/files/p17239293/s50860557/1bd4c21d-824a11b2-7cee1676-a5262fcf-94d431fc.jpg	no pneumothorax
MIMIC-CXR-JPG/2.0.0/files/p13342032/s57591238/fb0e8a56-6649854d-207b31d8-2b153049-441aeb82.jpg	no acute findings.
MIMIC-CXR-JPG/2.0.0/files/p11753351/s53973175/78691f88-984b7f7a-648f6bc1-d5dd3efc-9badddff.jpg	low lung volumes. no pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p19306644/s56329919/074c779a-0d456ff3-ca05b102-b4e8d36b-f8cb243c.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10585347/s50779726/3bd8c8df-ee49adac-50304c6d-6d3001a5-82a9a270.jpg	no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p12928318/s55953011/5badfdd9-b04bf6e9-6c8f2d0e-fbc9d798-e4b254a3.jpg	heart size and mediastinum are stable. port-a-cath catheter tip is at the level of superior svc. lungs are essentially clear. there is no pleural effusion or pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p15890451/s52144034/8d91019b-b1cee3c6-de9d6d62-3f7608b8-b75b2107.jpg	status post right upper lobectomy with left basilar opacity, which may represent atelectasis, pneumonia, or aspiration.
MIMIC-CXR-JPG/2.0.0/files/p16097925/s50732960/236a1eab-e9ed9249-b177694c-3cfb8d3f-26125880.jpg	diffuse bilateral airspace opacities are consistent with aspiration in the setting of massive upper gi bleed. monitoring and support devices are in the appropriate positions.
MIMIC-CXR-JPG/2.0.0/files/p19001926/s51026136/e98c2304-532bb52d-3cbe09dd-ad4438da-90df015a.jpg	endotracheal tube in standard position. consolidative opacities in the lung bases may reflect areas of infection. additional patchy opacities within the left lung and right upper lung field are concerning for additional sites of infection or aspiration. cardiomegaly with possible mild pulmonary vascular congestion.
MIMIC-CXR-JPG/2.0.0/files/p19575833/s54909865/95efe4b1-778960dd-e2984229-8a8f0a86-c1957068.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p16334734/s57687674/55c5e915-d63fa9d7-c4c38a80-d3578fc3-266a5c4c.jpg	increased opacity at the left lung base, concerning for pneumonia in the proper clinical setting.
MIMIC-CXR-JPG/2.0.0/files/p14944697/s54675870/ff227789-df655f92-d73b2ffb-a80f6f5b-70076187.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14634306/s53677592/29d5837a-4dc87534-6f166775-bda3059c-2a74ff02.jpg	ng tube tip is in the stomach. central venous line tip is in the proximal right atrium. no appreciable pneumothorax is seen. small bilateral pleural effusion is present.
MIMIC-CXR-JPG/2.0.0/files/p13954133/s50461527/95d3b6c2-54cfa6e2-c537b62e-ef37c75f-916857f9.jpg	apparent <num> mm nodule in the left peripheral mid lung, not definitively seen on prior, consider nonemergent chest ct to further assess. otherwise unremarkable.
MIMIC-CXR-JPG/2.0.0/files/p13383310/s55227511/4347632d-130ec954-632f7036-ffa8f88d-76f81a91.jpg	no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p15037339/s58651983/7103bc1b-f1af671e-77ee8be1-5c597199-a31db737.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p19230716/s56943510/dbda4432-8ac516cf-7cf12e2a-deb39454-cde97557.jpg	in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease or old tuberculous disease.
MIMIC-CXR-JPG/2.0.0/files/p16780133/s51712378/530e11d6-dfa5d6e4-a8f0d6fa-de20ba04-a63fbeb4.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17477807/s57996400/80ecf7f2-13cd0bad-146363bf-8d7d5ac7-3c2e0e70.jpg	ap chest compared to : a widespread global asymmetric predominantly interstitial pulmonary abnormality has developed since. it is accompanied by pulmonary and mediastinal vascular congestion, so it could be cardiogenic edema, but the heart has not increased in size. alternative possibilities are drug or transfusion reaction. dr was paged.
MIMIC-CXR-JPG/2.0.0/files/p13933813/s57810677/f3a9ab6f-85be7374-b73f8078-a92ab03f-9bf32161.jpg	no significant interval change when compared to the prior study. findings suggestive of pulmonary edema.
MIMIC-CXR-JPG/2.0.0/files/p18222456/s51222089/67ccc43a-a869b42c-44269fd5-6975cd06-6c06ed10.jpg	patchy opacities in the lung bases, more so on the left, may reflect areas of atelectasis but infection is difficult to exclude in the left lung base. possible trace right pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p16578063/s59602497/66f0e2a3-9290d095-9e4b766b-9b0134f6-a0b7e1ae.jpg	no evidence of acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15956776/s54819459/927358cb-a4db7ccc-dabf53bd-ec15d515-0ef274de.jpg	ap chest compared to : large right lower lobe pneumonia is slightly smaller today. previously airless left lower lobe is partially aerated, more likely resolving atelectasis than pneumonia. pulmonary and mediastinal vasculature are engorged, but there is no pulmonary edema, the heart is normal size. et tube and right internal jugular line are in standard placements respectively and two upper enteric tubes pass into the stomach and out of view. no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p12506591/s57707020/ab0e300b-1e59fb65-e0ac695c-9a467535-130e46b8.jpg	chronic lung changes without definite acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p18035751/s50613969/50398157-eb03ae75-799d03ae-cfd399d0-db9b3f00.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p13035993/s51744321/94635fc5-9895b266-92a66fe7-b126d655-8dd49332.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p13763648/s50857065/0f2300e4-c37561fb-a7b719e9-e4d0ac2c-54528e05.jpg	no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p11230841/s50434226/69633bcc-9052ef34-3192f8c0-01a713aa-c5746f97.jpg	clear lungs with no evidence of pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p10387770/s58071137/be61d297-18e294fa-c904e88a-849d2a9b-8324f81e.jpg	moderate right pleural effusion with overlying atelectasis, similar in extent as compared to the prior study; underlying consolidation not excluded.
MIMIC-CXR-JPG/2.0.0/files/p12406461/s51976390/4ab13540-f8742412-7ed217b5-e00f51ff-4fc9a702.jpg	right-sided central venous catheter terminates in the proximal to mid svc.
MIMIC-CXR-JPG/2.0.0/files/p14302681/s57393745/785a579b-044f1e2a-fa02d3dd-387904bb-64aee983.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11074330/s57830163/05a63fd1-acc6f8c3-bad6943c-0e4ae2f5-1d253d55.jpg	normal chest radiographs.
MIMIC-CXR-JPG/2.0.0/files/p18847905/s58311400/d53c787e-baf5a373-0584c752-f71ff377-af7b3648.jpg	no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p19059275/s57981151/a86a359a-155b1e90-a69dfbc4-aa7ac731-3b942107.jpg	unchanged appearance of the mild right apical hydropneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p11158097/s58807920/e645416c-764ecdfe-9c5bffd7-f679b14f-32523dee.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p12691278/s51960787/7067a7c7-4b2e8431-2188e89f-e561f949-b3fc823f.jpg	in comparison with the study , the right chest tube remains in place. there may be some increase in the degree of pleural if fusion, but no evidence of pneumothorax. otherwise, little change in the appearance of the heart and lungs.
MIMIC-CXR-JPG/2.0.0/files/p12299367/s51647737/f60c4f5f-abaf2f42-e1b0caec-7152ed08-8273d78b.jpg	no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p13504185/s58009641/c87debe4-c0d415b6-7a7ed324-313affce-f585838d.jpg	near complete opacification of the left hemi thorax with slight improvement and slight increase in aeration at the left upper lung.
MIMIC-CXR-JPG/2.0.0/files/p13044988/s58228598/b01e36d3-8c391380-b45e3d17-06124645-d9e05078.jpg	new right lower lobe atelectasis and bronchiolar inflammation with possible right lower lobe pneumonia. please correlate clinically. results were conveyed via telephone by dr to , np on at pm within <num> minutes of observation of findings.
MIMIC-CXR-JPG/2.0.0/files/p12331840/s58232768/a3c6433b-cdf05609-8b0feb27-59f5c6c4-c9668111.jpg	compared to the recent chest radiograph, a moderate sized left pleural effusion has increased in size with adjacent worsening left lower lobe atelectasis and or consolidation. similarly, a small to moderate right pleural effusion has also slightly increased in size. otherwise no relevant short interval change.
MIMIC-CXR-JPG/2.0.0/files/p10488031/s51799147/153ab00e-2a580347-4349b4ba-5f6a8ed7-d24f0701.jpg	slight interval worsening of the asymmetric interstitial pulmonary edema.
MIMIC-CXR-JPG/2.0.0/files/p15159308/s54504804/e8a56280-5220f146-58712401-8d8f2961-692beb46.jpg	improved vascular congestion
MIMIC-CXR-JPG/2.0.0/files/p10013569/s53407104/2bf1576a-cb1775c8-7235d1b9-91495ebc-f93e4a0a.jpg	enlarged cardiac silhouette and engorged pulmonary hila with pulmonary vascular congestion may be due to chf. right lower hemithorax opacity could be due to pleural effusions with overlying atelectasis and/or consolidation, elevation of the right hemidiaphragm. if patient able, dedicated pa and lateral views would be helpful for further evaluation.
MIMIC-CXR-JPG/2.0.0/files/p13603593/s59902163/7e16dbf9-07ff8840-ecad8bbe-aa8e5838-2544d271.jpg	left-sided port-a-cath terminates in the proximal to mid svc without evidence of pneumothorax. no priors available for comparison. there is volume loss of the left lung. left base opacity is seen which may be due to infection, aspiration, chronic change, related to patient's known lung cancer. left apical opacity could be due to pleural fluid, chronicity unknown. no focal consolidation, pleural effusion, or pneumothorax is seen on the right. cardiac silhouette is top-normal. left paratracheal opacity is seen, unclear whether this relates to patient's pulmonary malignancy. comparison with prior studies would be helpful for further assessment.
MIMIC-CXR-JPG/2.0.0/files/p13070030/s52601842/bde1eeac-71385956-e8396c7d-2506f3aa-a7a4f6dd.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15260348/s58851000/8e8b2085-2f14c7b5-fbace04d-666b6368-c64e7d00.jpg	bilateral effusions. no evidence of active disease, otherwise.
MIMIC-CXR-JPG/2.0.0/files/p18389073/s51466217/c4c505e9-bcc23c0c-2a70c3ae-aca1bf37-dc9cf112.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14181616/s53753978/9788f4b1-3b87d5b1-b1876925-7b906709-c7398553.jpg	there are persistent low lung volumes. right lower lobe collapse is unchanged. left lower lobe atelectasis has worsened. moderate vascular congestion is stable. there is no pneumothorax. if any there are small bilateral effusions. lines and tubes are in unchanged standard position
MIMIC-CXR-JPG/2.0.0/files/p14691641/s53998901/ce3d477d-fe08a6f7-74dc681f-7917760d-473bed72.jpg	ng tube has been advanced and appears appropriately placed. intra-aortic balloon pump was removed. swan-ganz catheter tip is in the main pulmonary artery. otherwise no significant change since :<num>
MIMIC-CXR-JPG/2.0.0/files/p18633403/s53485764/3665cacf-b81a7554-0f5820b2-a778ec4d-ed0bbe09.jpg	no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p10794198/s59886717/3421abb9-efeee7d5-bc628d37-fe6ee847-57a0c694.jpg	new diffuse interstitial opacities are consistent with clinical suspicion for fluid overload. accompanying asymmetrically distributed infrahilar alveolar opacities may reflect asymmetrical edema or coexisting aspiration. small bilateral pleural effusions are present, but there is no visible pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p11511428/s51928047/21d04b2f-7ec5b974-e63cf212-fc295e42-fd53b6b3.jpg	high riding endotracheal tube. advancement is recommended. appropriately positioned endogastric tube. extensive patchy consolidation within both lungs concerning for pneumonia/aspiration.
MIMIC-CXR-JPG/2.0.0/files/p14219992/s56635584/37ad5f2f-a433d054-1ae688ec-06af878e-f8c52dae.jpg	normal chest radiograph.
MIMIC-CXR-JPG/2.0.0/files/p15337600/s57131867/374abd84-ba4d38f6-2e4b781f-48cc9dc8-a99354e2.jpg	mild vascular congestion is unchanged. bilateral right perihilar and left lower lobe consolidations are grossly unchanged. cardiomegaly is a stable. tracheostomy tube is in standard position. pacer leads are in standard position. there is no pneumothorax or large pleural effusion
MIMIC-CXR-JPG/2.0.0/files/p10943603/s54991938/9c270069-9907cd80-8a02fa73-8a873508-c862676b.jpg	endotracheal and nasogastric tubes appropriately positioned. pulmonary edema with moderate right and tiny left pleural effusion. top normal heart size.
MIMIC-CXR-JPG/2.0.0/files/p16810793/s54262702/1229d83f-17761b7f-0dc0735f-3a44fc04-09021146.jpg	no acute cardiopulmonary process. persistently hyperinflated lungs compatible with emphysema.
MIMIC-CXR-JPG/2.0.0/files/p16335352/s52358693/c38928df-e368c14d-5533d9c8-002d48a2-d1a11faf.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10548962/s50950116/1fc3e6fd-156c619e-7a691dc8-16af5da5-e97cf6dc.jpg	as compared to the previous radiograph, no relevant change is seen. the lung volumes are low. borderline size of the cardiac silhouette. no larger pleural effusions. no pneumonia, no lymphadenopathy.
MIMIC-CXR-JPG/2.0.0/files/p12144619/s50013902/a3630df5-0a61d174-dbbd551e-7c999e67-dfcd99e5.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10011938/s54231050/de2c6694-9d5aa084-65ec7dd4-5d4c643c-08236601.jpg	no evidence of acute abnormalities. copd. peripheral lower lobe predominant reticulation is likely chronic.
MIMIC-CXR-JPG/2.0.0/files/p10786539/s55057779/02cbfc50-aa3287ff-fd6bb8ac-fbdd7710-3eb21bbc.jpg	lower lung volumes, without focal consolidation concerning for pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p13919890/s53699128/ebbabbfd-73b7a4f9-d740920b-b7ef3d0d-429c57c9.jpg	comparison to. status post bilateral thoracocentesis, the bilateral pleural effusions have minimally decreased in extent and severity but are still clearly visible. there is no evidence of pneumothorax. moderate cardiomegaly. sternotomy wires are in unchanged position. unchanged appearance of the lung parenchyma.
MIMIC-CXR-JPG/2.0.0/files/p16517702/s54820998/b94d6f54-5d1abef2-430e52cf-6ef5be94-40f44d56.jpg	no significant change compared with. no acute pulmonary process identified.
MIMIC-CXR-JPG/2.0.0/files/p10213338/s50988974/126a83fd-95dad2b1-6e3dcb66-3126546d-be2ac02a.jpg	in comparison with the study of , there is little overall change. mild hyperexpansion of the lungs again is seen with the cardiac silhouette at the upper limits of normal in size. no vascular congestion or acute focal pneumonia or pleural effusion.
MIMIC-CXR-JPG/2.0.0/files/p18033678/s58428009/3234a8e2-9ce6fadf-54bcfff9-7c97e851-990c9193.jpg	compared to chest radiographs. lung volumes are lower today, exaggerating top normal heart size. mediastinal widening suggests the new interstitial abnormality is mild pulmonary edema. no focal findings in the lungs to suggest pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p14857511/s52414859/97d1eef6-0335965c-32b24746-e0f155a6-10d8c3f2.jpg	moderate cardiomegaly with tiny left pleural effusion. hiatal hernia redemonstrated.
MIMIC-CXR-JPG/2.0.0/files/p12655574/s52537321/9537c965-60f7d581-8da7767e-0b52e5b6-4962ee19.jpg	mild-to-moderate interstitial pulmonary edema with associated small pleural effusions is new from prior exam. bibasilar opacities likely represent atelectasis or infection in the appropriate clinical setting. post diuresis cxr advised.
MIMIC-CXR-JPG/2.0.0/files/p19664531/s54975400/b3cef797-fdff4d6e-6b330be0-ac4bf090-3fc546e9.jpg	increased opacities at the lung bases could reflect aspiration, early infection or atelectasis.
MIMIC-CXR-JPG/2.0.0/files/p19000174/s58423485/9fc0ffbf-6f94b51a-d493187c-cc1ac1e7-b0b5ea87.jpg	no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14487480/s54409620/ccf292cc-26d6b173-107d3517-39adf90f-a1cf9463.jpg	minimal heterogeneous opacity in the left lower lobe along with a trace pleural effusion may represent aspiration. attention on followup imaging is recommended.
MIMIC-CXR-JPG/2.0.0/files/p18652728/s57751631/39e0b2b3-036387bb-40ce7e28-860d109f-0c22497f.jpg	no acute cardiopulmonary pathology.
MIMIC-CXR-JPG/2.0.0/files/p15737651/s52128232/1e9d7e42-9fb08fcd-3ec2c973-75fb7ed3-f35ca299.jpg	low lung volumes with possible minimal central pulmonary vascular engorgement.

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left upper extremity picc ends in the low svc.

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ap chest compared to , endotracheal tube, in standard placement, since the chin is flexed. right subclavian line has been repositioned, ends in the right atrium, and would need to be withdrawn approximately <num> cm to reposition it low in the svc, if desired. borderline cardiomegaly and mediastinal vascular engorgement are exaggerated by exceedingly low lung volumes and non-erect positioning. lungs are grossly clear. there is no pneumothorax or appreciable pleural effusion.

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interval worsening of near-complete opacification of the left hemithorax, likely related to large left-sided pleural effusion as seen on outside chest ct. new small right pleural effusion.

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no acute intrathoracic process.

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no acute cardiopulmonary process.

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increased interstitial markings, most significant at the left lung base, compatible with fibrotic changes likely secondary to underlying interstitial lung disease. pulmonary edema improved overnight. bilateral hilar prominence, which may be secondary to lymphadenopathy or pulmonary arterial enlargement and hypertension.

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no acute cardiopulmonary abnormality.

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unchanged moderate right pleural effusion.

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increased interstitial prominence, particularly in the right lung, since the prior study. airway inflammation or atypical infection could be considered in the appropriate clinical setting, although the appearance is not specific. no focal consolidation.

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collapse of the right middle lobe and right lower lobes is new. cardiomegaly cannot be evaluated. there is no evident pneumothorax. the left lung is grossly clear, aside from minimal retrocardiac atelectasis. right chest wall subcutaneous emphysema has improved

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no evidence of acute cardiopulmonary process.

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a comparison study of , the patient has taken a better inspiration. no evidence of pneumonia, vascular congestion, or pleural effusion. the configuration of the left port-a-cath is essentially unchanged.

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bilateral asymmetric pleural thickening with calcifications, suggestive of previous asbestos exposure. in this context, worsening basilar reticular opacities may reflect chronic interstitial lung disease with possible component of superimposed acute edema. follow-up chest radiograph could be performed after diuresis to further clarify this issue. high-resolution ct would also be helpful on a nonemergent basis to better characterize the patient's lung findings.

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vague opacity in the right mid-to-lower lung concerning for pneumonia. followup to resolution.

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no acute cardiopulmonary process. no evidence of free air beneath the diaphragms.

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probable left fibrothorax with chronic left pleural thickening, loculated fluid, and volume loss. small right pleural effusion.

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new right internal jugular central venous line nc in the right atrium at a level approximately <num> cm beyond the estimated location of the superior cavoatrial junction. no pneumothorax pleural effusion or mediastinal widening. mild cardiomegaly is exaggerated by low lung volumes. aside from calcified right upper lobe granuloma, the lungs are essentially clear. no pleural effusion or pneumothorax.

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compared to prior chest radiographs through. moderate to large bilateral pleural effusions and are larger and mild pulmonary edema has worsened. heart is normal size. t avr is grossly unchanged. no pneumothorax. atelectasis in the right upper lobe developed since and has not improved

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no acute cardiopulmonary process.

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interval worsening of cardiopulmonary findings

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hyperinflated lungs with evidence of biapical scarring, which most likely relate to copd. large hiatal hernia. no focal consolidation.

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et tube tip is approximately <num> cm above the carina.

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no evidence of pneumonia.

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in comparison with the study of , there again is extensive opacification in the right hilar region upper lung, consistent with the known malignant lesion and collapse of the right upper lobe. the primary tumor and mediastinal lymph nodes are much better appreciated on the previous ct study. there has been re-expansion of the remainder of the right lung with atelectatic streaks at the right base and. moderate subpulmonic pleural effusion is again seen. the left lung is essentially clear.

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no significant interval change.

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no acute cardiopulmonary pathology.

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increased opacification of the left hemithorax suggesting primarily increase in pleural effusion.

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small left pleural effusion. retrocardiac opacity may represent compressive atelectasis or aspiration, however pneumonia could be considered in the appropriate clinical setting.

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no pneumothorax

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no acute findings.

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low lung volumes. no pneumonia.

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no acute cardiopulmonary process.

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no acute cardiopulmonary abnormality.

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heart size and mediastinum are stable. port-a-cath catheter tip is at the level of superior svc. lungs are essentially clear. there is no pleural effusion or pneumothorax.

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status post right upper lobectomy with left basilar opacity, which may represent atelectasis, pneumonia, or aspiration.

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diffuse bilateral airspace opacities are consistent with aspiration in the setting of massive upper gi bleed. monitoring and support devices are in the appropriate positions.

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endotracheal tube in standard position. consolidative opacities in the lung bases may reflect areas of infection. additional patchy opacities within the left lung and right upper lung field are concerning for additional sites of infection or aspiration. cardiomegaly with possible mild pulmonary vascular congestion.

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no acute cardiopulmonary process.

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increased opacity at the left lung base, concerning for pneumonia in the proper clinical setting.

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no acute cardiopulmonary process.

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ng tube tip is in the stomach. central venous line tip is in the proximal right atrium. no appreciable pneumothorax is seen. small bilateral pleural effusion is present.

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apparent <num> mm nodule in the left peripheral mid lung, not definitively seen on prior, consider nonemergent chest ct to further assess. otherwise unremarkable.

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no evidence of pneumonia.

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no acute intrathoracic process.

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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease or old tuberculous disease.

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no acute cardiopulmonary process.

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ap chest compared to : a widespread global asymmetric predominantly interstitial pulmonary abnormality has developed since. it is accompanied by pulmonary and mediastinal vascular congestion, so it could be cardiogenic edema, but the heart has not increased in size. alternative possibilities are drug or transfusion reaction. dr was paged.

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no significant interval change when compared to the prior study. findings suggestive of pulmonary edema.

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patchy opacities in the lung bases, more so on the left, may reflect areas of atelectasis but infection is difficult to exclude in the left lung base. possible trace right pleural effusion.

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no evidence of acute cardiopulmonary process.

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ap chest compared to : large right lower lobe pneumonia is slightly smaller today. previously airless left lower lobe is partially aerated, more likely resolving atelectasis than pneumonia. pulmonary and mediastinal vasculature are engorged, but there is no pulmonary edema, the heart is normal size. et tube and right internal jugular line are in standard placements respectively and two upper enteric tubes pass into the stomach and out of view. no pneumothorax.

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chronic lung changes without definite acute cardiopulmonary process.

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no acute cardiopulmonary process.

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no acute intrathoracic process.

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no acute cardiopulmonary abnormality.

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clear lungs with no evidence of pneumonia.

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moderate right pleural effusion with overlying atelectasis, similar in extent as compared to the prior study; underlying consolidation not excluded.

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right-sided central venous catheter terminates in the proximal to mid svc.

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no acute cardiopulmonary process.

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normal chest radiographs.

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no acute cardiopulmonary abnormality.

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unchanged appearance of the mild right apical hydropneumothorax.

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no acute cardiopulmonary process.

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in comparison with the study , the right chest tube remains in place. there may be some increase in the degree of pleural if fusion, but no evidence of pneumothorax. otherwise, little change in the appearance of the heart and lungs.

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no acute cardiopulmonary abnormality.

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near complete opacification of the left hemi thorax with slight improvement and slight increase in aeration at the left upper lung.

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new right lower lobe atelectasis and bronchiolar inflammation with possible right lower lobe pneumonia. please correlate clinically. results were conveyed via telephone by dr to , np on at pm within <num> minutes of observation of findings.

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compared to the recent chest radiograph, a moderate sized left pleural effusion has increased in size with adjacent worsening left lower lobe atelectasis and or consolidation. similarly, a small to moderate right pleural effusion has also slightly increased in size. otherwise no relevant short interval change.

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slight interval worsening of the asymmetric interstitial pulmonary edema.

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improved vascular congestion

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enlarged cardiac silhouette and engorged pulmonary hila with pulmonary vascular congestion may be due to chf. right lower hemithorax opacity could be due to pleural effusions with overlying atelectasis and/or consolidation, elevation of the right hemidiaphragm. if patient able, dedicated pa and lateral views would be helpful for further evaluation.

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left-sided port-a-cath terminates in the proximal to mid svc without evidence of pneumothorax. no priors available for comparison. there is volume loss of the left lung. left base opacity is seen which may be due to infection, aspiration, chronic change, related to patient's known lung cancer. left apical opacity could be due to pleural fluid, chronicity unknown. no focal consolidation, pleural effusion, or pneumothorax is seen on the right. cardiac silhouette is top-normal. left paratracheal opacity is seen, unclear whether this relates to patient's pulmonary malignancy. comparison with prior studies would be helpful for further assessment.

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no acute cardiopulmonary process.

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bilateral effusions. no evidence of active disease, otherwise.

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no acute cardiopulmonary process.

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there are persistent low lung volumes. right lower lobe collapse is unchanged. left lower lobe atelectasis has worsened. moderate vascular congestion is stable. there is no pneumothorax. if any there are small bilateral effusions. lines and tubes are in unchanged standard position

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ng tube has been advanced and appears appropriately placed. intra-aortic balloon pump was removed. swan-ganz catheter tip is in the main pulmonary artery. otherwise no significant change since :<num>

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no acute intrathoracic process.

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new diffuse interstitial opacities are consistent with clinical suspicion for fluid overload. accompanying asymmetrically distributed infrahilar alveolar opacities may reflect asymmetrical edema or coexisting aspiration. small bilateral pleural effusions are present, but there is no visible pneumothorax.

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high riding endotracheal tube. advancement is recommended. appropriately positioned endogastric tube. extensive patchy consolidation within both lungs concerning for pneumonia/aspiration.

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normal chest radiograph.

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mild vascular congestion is unchanged. bilateral right perihilar and left lower lobe consolidations are grossly unchanged. cardiomegaly is a stable. tracheostomy tube is in standard position. pacer leads are in standard position. there is no pneumothorax or large pleural effusion

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endotracheal and nasogastric tubes appropriately positioned. pulmonary edema with moderate right and tiny left pleural effusion. top normal heart size.

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no acute cardiopulmonary process. persistently hyperinflated lungs compatible with emphysema.

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no acute cardiopulmonary process.

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as compared to the previous radiograph, no relevant change is seen. the lung volumes are low. borderline size of the cardiac silhouette. no larger pleural effusions. no pneumonia, no lymphadenopathy.

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no acute cardiopulmonary process.

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no evidence of acute abnormalities. copd. peripheral lower lobe predominant reticulation is likely chronic.

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lower lung volumes, without focal consolidation concerning for pneumonia.

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comparison to. status post bilateral thoracocentesis, the bilateral pleural effusions have minimally decreased in extent and severity but are still clearly visible. there is no evidence of pneumothorax. moderate cardiomegaly. sternotomy wires are in unchanged position. unchanged appearance of the lung parenchyma.

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no significant change compared with. no acute pulmonary process identified.

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in comparison with the study of , there is little overall change. mild hyperexpansion of the lungs again is seen with the cardiac silhouette at the upper limits of normal in size. no vascular congestion or acute focal pneumonia or pleural effusion.

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compared to chest radiographs. lung volumes are lower today, exaggerating top normal heart size. mediastinal widening suggests the new interstitial abnormality is mild pulmonary edema. no focal findings in the lungs to suggest pneumonia.

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moderate cardiomegaly with tiny left pleural effusion. hiatal hernia redemonstrated.

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mild-to-moderate interstitial pulmonary edema with associated small pleural effusions is new from prior exam. bibasilar opacities likely represent atelectasis or infection in the appropriate clinical setting. post diuresis cxr advised.

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increased opacities at the lung bases could reflect aspiration, early infection or atelectasis.

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no acute cardiopulmonary process.

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minimal heterogeneous opacity in the left lower lobe along with a trace pleural effusion may represent aspiration. attention on followup imaging is recommended.

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no acute cardiopulmonary pathology.

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low lung volumes with possible minimal central pulmonary vascular engorgement.