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endotracheal tube, nasogastric tube and left subclavian central line are unchanged in position. there is stable cardiac enlargement, which most likely represents cardiomegaly, although a pericardial effusion cannot be excluded. there is worsening bilateral airspace process suggestive of worsening edema, although diffuse pneumonia should also be considered. no large pneumothorax is appreciated.
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compared to prior chest radiographs through. left jugular catheters have been removed. moderate to large right pleural effusion has increased. left lower lobe atelectasis has worsened and there may be a new small left pleural effusion. moderate to severe enlargement of the cardiomediastinal silhouette is stable and pulmonary vasculature is engorged. there is no pneumothorax. right internal jugular line ends close to the superior cavoatrial junction.
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no pneumonia, edema, or effusion.
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small bilateral effusions. more focal opacities in the likely in the lingula and left lung base potentially atelectasis versus infection.
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no acute cardiopulmonary process.
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stable severe cardiomegaly from. stable large hiatal hernia from. bilateral layering pleural effusions and atelectasis. difficult to exclude infection in the right clinical setting.
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no acute cardiopulmonary process.
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termination point of left picc is still not identifiable. cross-sectional imaging would be the best option for definitively identifying the extent of the left picc. however, it terminates in at least the right atrium. findings were discussed by dr with dr by phone at on.
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no acute intrathoracic process.
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new predominantly perihilar confluent airspace opacities which may be reflective of pulmonary edema and/or multifocal infection.
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no acute cardiopulmonary process.
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bilateral patchy opacity lung bases, left greater than right, are concerning for pneumonia.
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no acute findings.
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ap chest compared to through : considerable consolidation has developed at both lung bases in the absence of edema or even much vascular engorgement in the upper lungs, and therefore could be considered pneumonia rather than asymmetric edema. severe cardiomegaly is chronic. right internal jugular line ends centrally. dr and i discussed these findings by telephone at
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no acute intrathoracic process. given multiple contours along the left heart border, consider oblique radiographs for further assessment. findings discussed with dr by dr at on by phone.
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moderately severe pulmonary edema is predominantly basal. cardiomegaly borderline. left pleural effusion is small. no pneumothorax.
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ap chest compared to , :<num> : mild pulmonary edema has worsened. consolidation in the right upper and lower lobes is stable or improved, unchanged at the left lung base. these findings raise concern for a concurrent multifocal pneumonia or atelectasis, perhaps related to aspiration. heart size mildly enlarged, unchanged. transvenous right atrial and ventricular pacer leads in standard placements. small bilateral pleural effusions unchanged. no pneumothorax. i discussed the findings by telephone with dr at
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as compared to the previous radiograph, the nasogastric tube has been slightly pulled back. the tip of the tube is located at the level of the gastroesophageal junction. partial left lower lobe atelectasis is improved. the bilateral apical opacities, however, are seen in almost unchanged manner. contrast material is seen in the stomach. no new focal parenchymal opacities. no larger pleural effusions. normal size of the cardiac silhouette
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normal radiograph of the chest.
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no acute cardiopulmonary process.
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no evidence of pneumonia.
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in comparison with the study of , there is still some is suggested increased opacification in the retrocardiac region. this could merely represent prominence of vessels, though the possibility of consolidation cannot be definitely excluded. if there is an appropriate clinical history to suggest possible central obstructing lesion leading to failure to clear a pneumonia, ct could be obtained. otherwise little change.
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normal chest radiograph.
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no significant change in size of left-sided pleural effusion. acute pulmonary edema.
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small bilateral pleural effusions, increased in size on the left, and unchanged on the right, with bibasilar atelectasis.
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in comparison with the earlier study of this date, there has been placement of a nasogastric tube that extends to the mid body of the stomach. the side port is probably close to the esophagogastric junction and the tube should be pushed forward at least <num> cm to be in more optimal position. there is increased haziness on the right, consistent with layering pleural effusion and compressive atelectasis at the base. retrocardiac opacification is consistent with volume loss in the left lower lobe and there is probably a small effusion on this side as well.
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probable new right lower lobe pneumonia/aspiration and persistent infection in the left lower lobe.
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patient has had median sternotomy. sternal wires are intact and aligned. mild cardiomegaly is more pronounced today than mild cardiomegaly is chronic. there is no pulmonary edema or focal pulmonary abnormality. no evidence of significant central adenopathy. mitral anulus is heavily calcified. multiple right posterior rib fractures are chronic and healed. a large lytic lesion is seen in the left scapula just inferior to the glenoid. patient has known myelofibrosis.
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no acute intrathoracic process. results were discussed over the telephone with dr by dr at on at time of initial review.
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no substantial interval change from the prior exam other than decreased lung volumes. continued small partially loculated right pleural effusion and trace left pleural effusion. right basilar patchy opacity may reflect atelectasis but aspiration or infection is not completely excluded.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
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moderate bilateral effusions with bibasilar atelectasis.
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since there been substantial changes: moderate enlargement of the cardiac silhouette is new, borderline interstitial edema has developed, small left pleural effusion is larger, and new heterogeneous opacification at the right base could be dependent atelectasis and edema. overall findings indicate cardiac decompensation. possibility of acute pulmonary embolism should be considered from a clinical standpoint, particular given.
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there are persistent low lung volumes. mild cardiomegaly is accentuated by the projection of the low lung volumes. there is mild vascular congestion. right lower lobe atelectasis is unchanged. left lower lobe atelectasis has minimally increased. there is no evident pneumothorax. if any there is a small right effusion.
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no acute cardiopulmonary abnormality.
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pa and lateral chest reviewed in the absence of prior chest radiographs: normal heart, lungs, hila, mediastinum and pleural surfaces. no evidence of tuberculosis active or prior.
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a gastrostomy tube is in place. lungs appear well inflated without evidence of focal airspace consolidation to suggest pneumonia or aspiration. no pleural effusions or pneumothoraces. no evidence of pulmonary edema. overall cardiac and mediastinal contours are stable. previously questioned abnormality at the right base likely reflected atelectasis which has resolved.
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as compared to the previous radiograph, the known pneumonia on the left has moderately increased in density. the left retrocardiac atelectasis is unchanged. the opacities in the right lower perihilar areas are constant in appearance. the monitoring and support devices are unchanged. no pneumothorax. unchanged size of the cardiac silhouette.
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no evidence of active or prior tuberculosis. no acute process.
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ap chest compared to : feeding tube has been removed. stomach is seriously distended with air and fluid. lungs are currently clear. transvenous right atrial and right ventricular pacer leads follow their expected courses. there is no pleural abnormality or evidence of central lymph node enlargement. right pic line ends in the mid svc. dr i discussed the findings by telephone.
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large left-sided basilar pneumothorax with possible rightward shift of the mediastinum raising concern for tension pneumothorax. gaseous distension of the stomach; consider placement of a ng tube for decompression.
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heart size and mediastinum are stable. lungs are clear. there is no pleural effusion or pneumothorax.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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in comparison with the study of , the pacing devices remain in place with no evidence of pneumothorax. there is stable enlargement of the cardiac silhouette with continued blunting of the right costophrenic angle. hyperexpansion of the lungs suggests underlying chronic pulmonary disease, but there is no evidence of acute focal pneumonia.
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persistent cardiomegaly. mild to moderate pulmonary vascular congestion. increase conspicuity of right lower lobe opacity worrisome for persistent pneumonia, with left base opacity also seen. no pneumothorax seen.
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retrocardiac opacity which may be due to a combination of atelectasis and pleural effusion, although a developing consolidation cannot be excluded. consider a lateral view for confirmation.
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no acute cardiopulmonary process.
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a pigtail catheter projects over the left hemithorax. no significant interval change in the appearance of the lung parenchyma. although no discrete pneumothorax is identified on the current examination, attention on follow-up imaging is recommended.
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in comparison with study of , there is little interval change. cardiac silhouette is within normal limits and there is moderate tortuosity of the descending aorta. no vascular congestion, pleural effusion, acute focal pneumonia, or pneumothorax.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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ap chest compared to : tip of the endotracheal tube is <num> cm or more from the carina. the chin is in neutral or elevated position. since more flexion of the head and neck is unlikely, the tube could be advanced another <num> cm for a more secured seating. based on the limitations of bedside radiographic technique of the patient of this size, i can say there is probably pulmonary and mediastinal vascular engorgement and moderate cardiomegaly, and in comparison to , conceivably mild interstitial edema, unless of course there are posteriorly layering pleural effusions, which would simulate edema. there is no pneumothorax. a feeding tube passes into the stomach and out of view.
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no acute cardiopulmonary abnormality. vertebral body height loss in the lower thoracic spine is uncommon for this patient's age, but unchanged compared to one month prior.
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post-pyloric tube folded upon itself in the third portion of the duodenum. results were discussed over the telephone with dr by on at at the time of initial review.
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similar chronic abnormalities with no definite superimposed acute process.
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as compared to the previous radiograph, no relevant change is seen. moderate cardiomegaly with signs of mild to moderate fluid overload but no evidence of pneumonia. minimal atelectasis at the right lung bases. no pleural effusions.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute findings. pacemaker appears in good position.
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left basilar opacity, which could be atelectasis, though in the proper clinical setting, pneumonia is a consideration. small bilateral pleural effusions and fluid layering along the right major fissure.
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improving left lower lobe opacity.
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no evidence of pneumothorax after transbronchial biopsy.
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no acute cardiopulmonary process.
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no evidence of focal consolidation.
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no acute cardiopulmonary process.
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subtle right middle lobe opacity concerning for early pneumonia.
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no acute cardiopulmonary process.
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mild hyperinflation. no evidence of acute cardiopulmonary disease.
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relative to prior examination, airspace opacification of the right mid and lower lobes is more conspicuous for which an infectious process is difficult to exclude. some fibrotic changes with architectural distortion and bilateral pleural thickening is stable in appearance relative to prior examination.
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right more than left lower lobe airspace opacities are new from prior exam, and while this appearance may represent asymmetric pulmonary edema, particularly in the setting of trace bilateral pleural effusions and mild cardiomegaly, this appearance could also represent multifocal infection. correlate with signs/symptoms and lab findings suggestive of infection. calcified pleural plaques are unchanged.
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right subclavian central venous catheter tip at the svc/right atrial junction.
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no acute intrathoracic abnormalities identified.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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as compared to the previous examination, the chest tube on the left is now attempt. there is a <num> mm left apical pneumothorax without evidence of tension. the position of the chest tube is unchanged. no other relevant changes as compared to the previous image.
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compared to chest radiographs through. symmetric bibasilar interstitial abnormality is new. because of its distribution edema is the most likely explanation. however heart size is normal and mediastinal veins are not dilated so the explanation is not necessarily cardiogenic. clinical correlation advised. right subclavian infusion port ends in the low svc.
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no radiographic evidence of an acute cardiopulmonary process.
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no acute findings. if there is strong clinical concern for rib fracture, recommend dedicated rib series to further assess.
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no evidence of pneumonia. no acute cardiopulmonary process.
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increased bilateral mid to lower lung opacities are likely worsening pulmonary edema. superimposed right lower lung consolidation cannot be ruled out.
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MIMIC-CXR-JPG/2.0.0/files/p15448674/s58636727/a71770dc-5fee9b66-2c37006e-f2029502-5a37201b.jpg
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mild-to-moderate interstitial abnormality most suggestive of pulmonary vascular congestion.
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as compared to radiograph, a subdiaphragmatic lucency has decreased in extent and likely reflects resolving pneumoperitoneum. exam is otherwise remarkable for slight increase in size of small to moderate left pleural effusion with adjacent left basilar atelectasis. small right pleural effusion persists.
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slight improvement in vascular congestion, with continued low lung volumes and small right pleural effusion.
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MIMIC-CXR-JPG/2.0.0/files/p11945540/s55121643/26cbabee-163ef10c-304aff64-6226e36f-df9ab576.jpg
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no acute cardiopulmonary process.
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right lower lobe pneumonia.
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normal postoperative cardiomediastinal silhouette. tiny right apical pneumothorax unchanged. no left pneumothorax or appreciable pleural effusion. no pulmonary edema. in standard placements and/or unchanged are the et tube, midline and left pleural drainage catheters, transvenous right atrial and right ventricular pacer leads and a nasogastric drainage tube. the swan-ganz catheter loop projects main pulmonary artery, but the tip could be in the left pulmonary artery as well.
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chronic fine reticular opacities of the lateral lung bases; otherwise clear lungs.
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lungs are well expanded and clear. heart is borderline enlarged but the pulmonary vasculature is unremarkable. incidental note is made of an azygos fissure. no evidence of central lymph node enlargement. no pleural abnormality.
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moderate left pleural effusion, which has minimally increased from the prior study. bibasilar atelectasis, left greater than right.
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resolution of left lower lobe pneumonia.
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no evidence of acute cardiopulmonary process or displaced rib fracture. no pneumothorax.
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moderate right pleural effusion is larger than on. severe enlargement of the cardiac silhouette is stable. pulmonary vascular engorgement is mild and there is no edema. transvenous right atrioventricular pacer leads are in standard placements.
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moderate bilateral pleural effusions with overlying atelectasis. pulmonary vascular congestion. mild cardiomegaly.
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no acute intrathoracic process.
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no acute cardiopulmoanry process.
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subtle opacification within the left lower lobe, representing an early/developing pneumonia. recommendation(s): a chest radiograph in <num> weeks following treatment is recommended to assess resolution.
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no acute cardiopulmonary process. a nodular opacity projecting over the anterior right sixth rib is new compared to , and likely represents a nipple shadow.
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MIMIC-CXR-JPG/2.0.0/files/p12000432/s51638543/da601fa1-b8025621-cc16bfa6-db14f757-cac7f7ae.jpg
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possible minimal interstitial edema. otherwise, no acute cardiopulmonary process.
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