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MIMIC-CXR-JPG/2.0.0/files/p13814783/s55478505/d9a3905a-2e45e1a1-cf74d466-336b533f-acee8fb9.jpg
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retrocardiac consolidation is unchanged from. pa and lateral would be helpful for further evaluation.
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no radiographic evidence for acute cardiopulmonary or chronic granulomatous disease.
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in comparison with the study of , the apical pneumothorax appears to be slightly smaller. chest tube remains in place. otherwise little change.
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mild pulmonary edema, no pneumonia.
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no acute cardiopulmonary process.
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no pneumonia. trachea is deviated to the right with prominent soft tissue in the upper mediastinum possibly related to position but a substernal goiter is possible. recommend comparison with prior chest radiographs if available, and if not follow up pa and lateral chest x-ray in months. an email was sent to the ed qa nurses regarding the recommendation for follow up.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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right ij catheter is <num> cm beyond the superior cavoatrial junction. subsequent image demonstrates an appropriate position of the catheter in the mid-svc.
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as compared to the previous radiograph, the appearance of the cardiac silhouette, the monitoring and support devices, the retrocardiac atelectasis and the remaining lung parenchyma are unchanged. only the right lung base has slightly increased in radio transparency, reflecting improved ventilation. no other relevant changes.
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standard position of monitoring and support devices.
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no evidence of acute cardiopulmonary abnormality. bibasilar reticular interstitial markings are improved, possibly reflecting nsip response to treatment.
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MIMIC-CXR-JPG/2.0.0/files/p11123733/s55208039/32af453c-cc38b779-3110ed99-96be9cc6-01a2f654.jpg
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mild bibasilar atelectasis and small bilateral pleural effusions. copd.
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MIMIC-CXR-JPG/2.0.0/files/p17821017/s54113432/e304c64c-c64edf6f-81e81100-ffa14746-a06e30c5.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p10675468/s50947004/e367ad8d-48191c0d-05e10c13-83db1618-033c0014.jpg
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no acute cardiopulmonary process.
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no pneumonia, edema, or effusion.
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decreasing but persisting pulmonary edema.
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as compared to radiograph, endotracheal tube is been advanced slightly, now terminating <num> cm above the carina. lung volumes are lower, resulting in crowding of bronchovascular structures. a subtle patchy opacity at the left lung base could reflect patchy atelectasis, aspiration, or an early focus of pneumonia. short-term followup radiographs may be helpful in this regard. note is also made of gastric distension in the imaged portion of the upper abdomen.
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mild pulmonary vascular congestion is new since the prior chest radiograph.
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ap chest compared to : right middle lobe is collapsed and consolidation at the base of the right lung has not improved since. findings point to pneumonia or bronchial obstruction, perhaps due to aspiration. right upper lung and left lung are clear. heart size is normal. there is no pleural effusion.
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no acute cardiopulmonary process. no displaced rib fracture is seen. however, the pa and lateral chest radiograph is insensitive for the detection of subtle rib fractures. if there is continued clinical concern, consider obtaining dedicated rib series.
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no acute cardiopulmonary process.
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the heart remains stably enlarged. the aorta is prominent and unfolded. inspiration on the frontal view has improved, although on the lateral view, there is still substantial vascular crowding. patchy opacity in the retrocardiac region could reflect atelectasis although an early infectious process is possible in the correct clinical setting. clinical correlation is recommended. no pulmonary edema or pneumothorax. no large effusions.
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no evidence of acute cardiopulmonary disease.
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MIMIC-CXR-JPG/2.0.0/files/p10455855/s51125904/41ad0047-5ca8758d-83007b42-0d85323e-870f9c85.jpg
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persistent bibasilar opacities may represent combination of pleural effusions and atelectasis, underlying consolidation not entirely excluded. enlargement of the cardiac silhouette. pulmonary edema, somewhat improved from the prior study.
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compared to chest radiographs since , most recently. previous nodular opacities in the upper lungs have largely cleared. this could of been either rapidly responsive infection or asymmetric edema. the only focal pulmonary abnormality today's consolidation at the right lung base which could be either pneumonia or atelectasis, increased since. previous pulmonary edema has almost resolved although pulmonary vasculature is engorged, while mediastinal caliber has decreased since. heart size is top-normal. pleural effusions small if any. no pneumothorax.
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resolution of previous pneumonia.
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no acute cardiopulmonary process.
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no displaced rib fracture identified. dedicated rib films with the marker over the point seen to be considered if clinical suspicion for rib fractures remains high.
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hyperinflated lungs without radiographic evidence for acute cardiopulmonary process.
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no evidence of pneumonia.
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ap chest compared to : tip of the right subclavian line ends in the mid-to-low svc. no pneumothorax, pleural effusion or mediastinal widening. heart size normal. lungs grossly clear. healed left lower lateral rib fractures noted.
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probable multilobar pneumonia of the right lung, stable from two days ago and much improved from. a focal remaining component of pneumonia versus pleural effusion tracking into the fissures on the right. oblique views may help differentiate the two possibilities. stable congestive heart failure.
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progressive right middle lobe collapse in a patient with a known infiltrative right middle lobe lung carcinoma increasing right hilar opacity secondary to probable superimposed infection, better characterized on concurrent ct. small right pleural effusion
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no radiographic evidence for acute cardiopulmonary process.
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right pleural effusion and multi focal pneumonia.
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new moderate subcutaneous emphysema and small pneumomediastinum. lines and tubes remain in satisfactory position. no other significant interval change from the study of <num> day prior.
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no acute cardiopulmonary process.
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comparison to. the patient has received the intra-aortic balloon pump. the tip of the pump projects approximately <num> cm be low the upper most portion of the aortic arch. the patient also has received a swan-ganz catheter. the tip is in the left pulmonary artery. in the interval, the patient has developed a fairly asymmetrical pulmonary edema, predominating in the right mid and low lung zones. the stomach is overinflated, so that insertion of a nasogastric tube could be considered. moderate cardiomegaly.
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comparison to. no relevant change. stable borderline size of the cardiac silhouette. stable appearance of non recent parenchymal opacities predominating in the upper lobe bases and in the perihilar lung areas. no new focal parenchymal opacities. no larger pleural effusions. no pulmonary edema. stable correct position of the left picc line.
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in comparison with the study of , there is increasing opacification at the left base. this is consistent with developing pneumonia. blunting of the costophrenic angle could reflect a small pleural effusion. mild atelectatic changes are seen on the right. an
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stable mild cardiomegaly. left base opacity as described above and similar to prior. suspect underlying effusion and atelecatsis although infection is not excluded. consider pa and lateral views to further charcterize when patient is amenable.
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no acute cardiopulmonary process. no evidence of pneumonia, pneumothorax, or pneumomediastinum.
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MIMIC-CXR-JPG/2.0.0/files/p13327132/s58688986/75cdfc13-7c2e396a-b38759a5-0195602c-3c4e8236.jpg
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no acute cardiopulmonary process. mild emphysematous changes.
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no pneumothorax. these findings were discussed with by via telephone on , at , at time of discovery.
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no acute cardiopulmonary process. the mildly enlarged hilar lymph nodes seen on the chest ct done today are less well seen on this radiograph.
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previously seen left apical pneumothorax, if anything is insignificant.
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no evidence of pulmonary nodule on this radiograph.
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with the study of , the previously suggested nodular opacification at the left base is not identified. there is no evidence of pneumonia, vascular congestion, or pleural effusion. no evidence of old tuberculous disease.
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no acute cardiopulmonary process.
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interval removal of bilateral pigtail pleural catheters. on the left, there may be a small loculated left basilar pneumothorax but this can be further assessed on followup imaging. no right pneumothorax is seen. there is streaky patchy opacity in the left mid peripheral lung as well as more focal opacity in the right mid-to-lower lung which may represent a combination of atelectasis, scarring and/or loculated pleural fluid. clinical correlation is advised. no evidence of pulmonary edema. overall cardiac and mediastinal contours are stable. lungs are hyperinflated consistent with underlying emphysema, and there is blunting of both costophrenic angles likely reflecting chronic pleural thickening or small effusions. prominent pulmonary arteries raise the possibility of underlying pulmonary arterial hypertension.
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p17079101/s58066330/b6d980d3-b586c166-d29fa9de-6a7335ae-35148194.jpg
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increased opacification of the right lung, likely a combination of pleural fluid and underlying atelectasis. given the mild pulmonary vascular congestion, this may represent fluid overload but given the recent history of trauma, injury is not excluded. if there is clinical concern for trauma, a non-contrast chest ct would be recommended for further evaluation.
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no acute intrathoracic process identified. no displaced rib fracture is identified.
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right middle lobe pneumonia.
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diffuse bilateral abnormality in the lungs, previously characterized by ct. no definite acute superimposed process, although subtle change could easily be obscured.
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as compared to the previous radiograph, there is a minimal increase in extent of the pre-existing left pleural effusion. the consolidation at the bases of the right upper lobe is constant. no pulmonary edema. moderate cardiomegaly persists. no pneumothorax.
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interval placement of dobhoff tube that terminates within the stomach. incidental finding of a intragastric tooth. stable cardiopulmonary findings when compared to the earlier study on.
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MIMIC-CXR-JPG/2.0.0/files/p16207932/s53222869/4837215d-a6d9697f-5ae0e3e4-29d067c6-8f4a5f94.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p12385889/s59314903/793ef587-c5074621-b0be61ef-2d60eb24-c22b1dee.jpg
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no evidence of infection or malignancy; however, pneumonia can be radiographically occult on plain film. if there is sufficient clinical concern, ct of the chest is recommended for better assessment of possible infection.
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moderate right pleural effusion has increased, and moderately severe right basal unchanged. pulmonary vascular engorgement has improved since , but mediastinal widening suggests persistent distension of mediastinal veins. moderate enlargement of the cardiac silhouette which had increased from until is unchanged, due to increasing moderate cardiomegaly and/or pericardial effusion. no pneumothorax. left pic line ends outside the chest in the left axilla, as before.
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no acute cardiopulmonary abnormality.
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MIMIC-CXR-JPG/2.0.0/files/p15311917/s59199089/c8b512ed-a1007374-5c37d9ce-4f00a131-64d2655c.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p10836389/s55469186/41e65d86-038d122f-7b36e54f-c2dccced-0617b089.jpg
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no acute cardiopulmonary abnormality.
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slight improvement in pulmonary edema
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et tube tip is <num> cm above the carinal. left internal jugular line tip at the level of mid svc. low right <num> chest tubes are in unchanged location. there is extensive subcutaneous air that appears to be a similar to previous examinations. right lung consolidations are unchanged.
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p15693424/s52911013/0e7e2a2c-122684d7-00f615b7-9c6de326-36100818.jpg
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as compared to the previous image, the patient has received a left pectoral pacemaker. <num> lead projects over the right atrium and <num> over the right ventricle. no pneumothorax or other complications. no pulmonary edema.
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no previous images. cardiac silhouette is within normal limits and there is no vascular congestion, pleural effusion, or acute focal pneumonia.
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compared to the chest radiograph. lung volumes are lower exaggerating mild pulmonary vascular congestion and moderate cardiomegaly. mediastinal veins are engorged. pulmonary edema is probably not present and pleural effusions are small if any. no focal pulmonary abnormality appreciated.
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no acute cardiopulmonary process.
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in comparison to exam, small pleural effusions and mild interstitial pulmonary edema has resolved. mild perihilar vascular congestion persists.
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enteric tube coiled in the stomach, tip in the proximal stomach. worsened right basilar consolidation, consider infection.
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in comparison with the study of , there is little overall change. again there is huge enlargement of the cardiac silhouette with relatively mild pulmonary vascular congestion, a discordance that suggests underlying cardiomyopathy. lvad remains in place. given the size of the heart, it it would be impossible to exclude the possibility of superimposed pneumonia in the appropriate clinical setting, especially in the absence of a lateral view.
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p14006201/s51041650/0545f4f6-3fb49fda-f51d7ca1-4c49f45c-43ace359.jpg
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heart size top-normal.
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no acute cardiopulmonary process.
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subtle left base opacity is felt to more likely be due to atelectasis but infection.
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no acute cardiopulmonary process.
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small residual lingular opacity could represent sequela of prior infection although superimposed component of acute infection is hard to exclude.
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increased bibasilar atelectasis and mild pulmonary vascular congestion.
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MIMIC-CXR-JPG/2.0.0/files/p15541469/s59150757/b103abaf-67328087-7e1f38c3-7f24fda9-870fa9d8.jpg
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as compared to the previous radiograph, pre-existing opacities at the lung bases have completely cleared. no new opacities are visualized. no pneumothorax. hypoplastic first rib.
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MIMIC-CXR-JPG/2.0.0/files/p11327070/s50001886/55d370ca-bdd36532-d321a013-50ad1c19-867da3cf.jpg
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small bilateral pleural effusions. no focal consolidation or pneumothorax.
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MIMIC-CXR-JPG/2.0.0/files/p17347108/s58568383/a0bba0b4-4cf480c8-b9bbdf5f-91bea027-e6cf0f62.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p10725976/s56830159/b5fe07af-1edbba06-4314427c-dfd79167-6c1c8239.jpg
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heart size is top-normal. mediastinum is unremarkable. lungs are clear. there is no pleural effusion or pneumothorax
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MIMIC-CXR-JPG/2.0.0/files/p13312252/s50891249/a817f8e4-cb9834b1-66b610fd-9aeae9b6-0b5e7cde.jpg
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interval retraction of the endotracheal tube which now projects <num> cm from the carina. no other significant interval change from the prior study.
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MIMIC-CXR-JPG/2.0.0/files/p14923390/s52892057/5bfce132-94a9b5d0-10240b5e-d26e05f3-b29955fe.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p13790147/s56605219/8a54e811-76a51cbd-0660347b-bdfaf717-eaba9450.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p19765303/s53283325/a500a63c-aa97da86-a3e48cbc-04d09c87-f8c3cccf.jpg
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cardiomegaly with mild pulmonary edema. likely small bilateral pleural effusions.
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MIMIC-CXR-JPG/2.0.0/files/p17516297/s54220732/a6cb29a7-ef301599-9aa1aa55-3356028d-484235d4.jpg
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<num>) no acute cardiopulmonary process. <num>) new compression fracture of approximately the t<num> vertebrae (since ).
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MIMIC-CXR-JPG/2.0.0/files/p15173539/s50829111/9709ad60-53c76904-7245d899-fe17edff-6c4e0f8c.jpg
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pa and lateral chest compared to : aside from a handful of calcified granulomas, lungs are clear. cardiomediastinal and hilar silhouettes are unremarkable aside from probable right hilar lymph nodes. there are no findings to suggest active infection or malignancy. heart is normal size, and there is no pleural abnormality.
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MIMIC-CXR-JPG/2.0.0/files/p18426683/s55641005/cf5924ba-ae1a57e6-7c12714d-c6cdff40-a8a7c6e2.jpg
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in comparison with the study of , there is now a stent within the left mainstem bronchus. the monitoring and support devices are essentially unchanged. little overall change in the appearance of the heart and lungs.
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MIMIC-CXR-JPG/2.0.0/files/p16568324/s52571064/53af3892-162ccc59-42c6e972-e0eb2559-d7760502.jpg
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enteric tube tip remains in the distal esophagus and should be advanced for optimal positioning.
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MIMIC-CXR-JPG/2.0.0/files/p14600571/s59007065/86c9d0eb-b2e8df22-7de70cf2-2957a868-c60a4361.jpg
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in comparison with the study of , the patient has undergone a segmentectomy with left chest tube in place. no evidence of pneumothorax. there is some subcutaneous gas along the left lower neck. no evidence of acute pneumonia or vascular congestion. no pleural effusion identified.
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MIMIC-CXR-JPG/2.0.0/files/p11751302/s59604412/b14aa074-e04b52d7-fdd11f67-154c99b4-ae414eb4.jpg
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ng tube with tip in the gastric body, side port in the region of the ge junction.
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MIMIC-CXR-JPG/2.0.0/files/p13166511/s55648381/f21548cd-a7e7b70e-a2a9aceb-8a3b0ede-f7091b12.jpg
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no evidence of pneumonia.
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MIMIC-CXR-JPG/2.0.0/files/p15556497/s59707071/011c40f4-a4ddd770-3859f2d3-59b67e4c-e958b1aa.jpg
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in comparison with the study , there continued low lung volumes that accentuate the transverse diameter of the enlarged heart. continued pulmonary edema with left pleural effusion and volume loss in the left lower lobe. multiple rib fractures are again seen.
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MIMIC-CXR-JPG/2.0.0/files/p17058654/s59172992/0a591834-7d6f504f-60a60c94-5ff8906d-1d13a003.jpg
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as compared to the previous radiograph, the sternal fixation devices are in unchanged position. unchanged monitoring and support devices. the lung volumes have decreased, with the resulting increase in profusion and severity of the pre-existing micronodules and vascular changes. increase in extent of a retrocardiac atelectasis. no larger pleural effusions.
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MIMIC-CXR-JPG/2.0.0/files/p11619702/s59251333/845c4e74-9837c4c5-622a4cea-70e3902e-4406ae9b.jpg
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hyperinflation without focal consolidation.
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MIMIC-CXR-JPG/2.0.0/files/p14903003/s56295240/257071da-8377e841-d945d669-2a0d92f5-2a8c6cd5.jpg
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left basilar opacification suggesting pleural effusion in combination with parenchymal opacification and elevation of the left hemidiaphragm. parenchymal opacification is compatible with associated atelectasis, although it is difficult to entirely exclude a coinciding infectious process.
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