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MIMIC-CXR-JPG/2.0.0/files/p11424643/s59231627/07d9cf70-201000ef-7ec884b2-938d33b2-3ac6c7e3.jpg
mild basilar atelectasis without definite focal consolidation.
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no acute cardiac or pulmonary findings.
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no acute cardiopulmonary process.
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mild pulmonary edema.
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interval increase in now moderate to large right pleural effusion with overlying atelectasis. central pulmonary vascular engorgement.
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lungs are fully expanded and clear. normal cardiomediastinal and hilar silhouettes and pleural surfaces. right central venous infusion port ends in the region of the superior cavoatrial junction. left subclavian line ends in the upper to mid svc.
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the lung volumes have decreased. moderate cardiomegaly. small left pleural effusion with small retrocardiac atelectasis. unchanged position of the right-sided port-a-cath. a new right internal jugular vein catheter has a normal course and the tip projects over the cavoatrial junction. no evidence of complications, notably no pneumothorax.
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no acute cardiopulmonary process.
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patchy opacities within the peripheral aspect of the right mid lung field, likely the right upper and middle lobes, as well as the left lung base, in a distribution similar to that seen on the prior radiographs from , but somewhat improved. while these findings could reflect recurrent pneumonia, other processes including inflammatory processes should be considered. followup radiographs after treatment are recommended, and if the findings persist, a chest ct is suggested.
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probable right lung pneumonia, possibly related aspiration.
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no focal consolidation. chronic scarring bilaterally. mild pulmonary vascular congestion.
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no acute cardiopulmonary process. unchanged expected location of left-sided port-a-cath.
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there has been enough improvement in the severe infiltrative pulmonary abnormality in the lungs to make it apparent that there are many lung nodules from <num> mm in diameter. some nodules may have been present since , obscured by the generalize process, but i suspect more nodules have formed. this points to disseminated infection. it would be very helpful to do a ct scan. the heart is normal size. endotracheal tube ends in at the carina an should be withdrawn <num> cm. house staff notified. nasogastric tube ends in the stomach. pleural effusion is small on the right. no pneumothorax.
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small left lower lobe consolidation likely represents pneumonia.
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no definite acute cardiopulmonary process. age indeterminant mild vertebral body height loss in the mid-to-lower thoracic spine. clinical correlation suggested.
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in comparison with the study of , the right ij sheath has been removed. there is enlargement of the cardiac silhouette with pulmonary edema and bilateral pleural effusions with compressive atelectasis at the bases. given the extensive pulmonary changes, it would be very difficult to exclude superimposed pneumonia in the appropriate clinical setting, especially in the absence of a lateral view.
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no acute intrathoracic process.
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no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p15568681/s52185482/5e54be80-f4250e43-e72a5b38-8485384b-789ffdda.jpg
no evidence of acute cardiopulmonary abnormalities.
MIMIC-CXR-JPG/2.0.0/files/p13117621/s55157459/4d4974ba-0afe2a44-39236811-c366322c-1b6e3e29.jpg
no acute cardiopulmonary process.
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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease. no pneumonia, vascular congestion, or pleural effusion.
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no evidence of acute cardiopulmonary disease.
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no acute intrathoracic abnormality.
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possible component of fluid overload which is difficult to assess given large body habitus.
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nodular opacities at the lung bases bilaterally, which likely represent nipple shadows. in addition, there is an opacity overlying the right anterior seventh rib, which may represent callus at a rib fracture. however, <num> are more pulmonary nodules cannot be excluded in this patient with emphysema. multiple stable compression deformities within the thoracic spine.
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new left lower lobe opacification is concerning for pneumonia with parapneumonic pleural effusion.
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chest findings within normal limits, thus no evidence of acute pneumonic infiltrate in this -year-old female patient with cough, also developing fever.
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ng tube tip in the stomach
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there continued to be bilateral airspace opacities which could represent pneumonia or atelectasis. there are bilateral pleural effusions. the contours of the effusions suggest that the fluid may be somewhat loculated, particularly on the right. there is no evidence of pulmonary edema. overall cardiac and mediastinal contours are unchanged. a portion of a biliary stent is again visualized. overall, there does not appear to be any significant interval change. no pneumothorax.
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no definite acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p12574181/s59618294/0547fcee-c8833eed-1aa276a7-d45a9ef3-966b2a9c.jpg
no acute cardiopulmonary process.
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compared to prior chest radiographs through. lungs fully expanded and clear. mild cardiomegaly improved since. no pneumothorax pleural effusion or pulmonary vascular abnormality. right transjugular swan-ganz catheter ends in the lower right descending pulmonary artery. left trans subclavian right atrial, left ventricular pacer and right ventricular pacer defibrillator leads in standard placements.
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no evidence of acute cardiopulmonary disease.
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cardiomegaly without acute intrathoracic process.
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no acute pulmonary process identified. no focal infiltrate to suggest pneumonia.
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no acute cardiopulmonary abnormality.
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no evidence of a pneumothorax status post lead revision.
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no acute cardiopulmonary process.
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findings suggest mild fluid overload.
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pneumonia in the superior segment of left lower lobe.
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mild cardiomegaly, interstitial edema, small right pleural effusion. ill-defined opacity in the right lung base is concerning for pneumonia. followup to resolution is advised.
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no pneumothorax. the appearance of the known left upper lobe mass is not significantly changed since. small left pleural effusion.
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ap chest compared to :<num> feeding tube, wire stylet removed, ends in the upper stomach. mild cardiomegaly stable. lower lungs clear. lung apices excluded, but sufficiently imaged to show bronchiectasis and pleural thickening, most likely scarring from previous tuberculosis.
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right lower lung pneumonia. left picc line terminates in the right atrium. recommend pulling back <num> cm to position it at the cavoatrial junction.
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no acute cardiopulmonary process. bilateral reticular opacities are noted, most notably in the right upper lung, unchanged from prior exams and consistent with known emphysematous changes
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no acute intrathoracic process.
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ap chest provided for review on : large right pleural effusion, moderate left pleural effusion, severe pulmonary edema and mediastinal vascular engorgement are all unchanged. the heart is moderately enlarged. et tube in standard placement. nasogastric tube passes below the diaphragm and out of view. right subclavian or picc line ends just before the junction of the brachiocephalic veins.
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as compared to the previous radiograph, no relevant change is seen. larger right hilar lesion with interstitial markings, suggesting local lymph an juices. decreased left hemithoracic volume with a small pleural effusion. enlargement of the aortopulmonary window, likely caused by component of the known mass. unchanged appearance of the sternal wires.
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no relevant change as compared to the previous image. minimal atelectasis at the right lung bases. no evidence of pulmonary fibrosis. borderline size of the cardiac silhouette with mild elongation of the descending aorta. no pneumonia. unchanged course of the pacemaker leads.
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low lung volumes with persistent small to moderate bilateral pleural effusions and severe bibasilar atelectasis.
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increasing pulmonary vascular congestion without pulmonary edema. bibasilar linear atelectasis. stable position of et tube and enteric tube. possible left upper lung pleural-based calcifications. correlate with prior radiographs. if prior radiographs are not available, we recommend non-emergent chest ct for further evaluation.
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heart size is within normal limits. there are low lung volumes with atelectasis at the lung bases. no overt pulmonary edema or focal consolidation is seen. there are no pneumothoraces. there has been no interval change.
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no signs of pneumonia. emphysema again noted.
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mild peribronchial wall thickening in streaky perihilar opacities, which can be seen in the setting of reactive or small airway disease. no focal consolidation to suggest bacterial pneumonia. no appreciable pneumothorax, though the left lung apex is obscured by an overlying lead.
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no acute cardiopulmonary process.
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in comparison with the study of , the cardiac silhouette remains at the upper limits of normal in size or slightly enlarged. there is no definite pulmonary vascular congestion or acute focal pneumonia. atelectatic changes with possible small effusion appear more prominent at the left base on the current study.
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no evidence of acute cardiopulmonary disease.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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worsening right basilar opacity and possible new right apical opacity concerning for worsening infection. aspiration is not excluded. small right pleural effusion is unchanged from.
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diffuse increased vascular markings likely reflects chf. low inspiratory volumes noted.
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findings suggestive of a congestive failure with vascular congestion, bilateral pleural effusions and cardiomegaly. interval compression deformity of a mid thoracic vertebral body age indeterminate but new since.
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confluent regions consolidation and the left lung compatible with pneumonia in the proper clinical setting. recommend repeat after treatment to document resolution and to exclude underlying mass lesion.
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persistent collapse of the left lower lobe. small left pleural effusion is unchanged. previously seen left pneumothorax is no evident. aeration of the right lung has improved. lines and tubes are in unchanged standard position. cardiomediastinal structures are shifted to the left as before. cardiac size is normal
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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease. continued chronic elevation of the left hemidiaphragmatic contour. however, no evidence of cardiomegaly, vascular congestion, pleural effusion, or acute focal pneumonia.
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in comparison with study of , the cardiac silhouette remains within normal limits. no vascular congestion or acute focal pneumonia. mild blunting of the right costophrenic angle, which could represent pleural scarring.
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comparison to. <num> images both show the tip of the feeding tube in the middle parts of the esophagus. no complications, notably no pneumothorax. the appearance of the remaining lung parenchyma is unchanged.
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moderate to severe cardiomegaly without pulmonary edema or pneumonia.
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no acute cardiopulmonary process.
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no significant interval change. no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute findings in the chest.
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ap chest compared to : severe infiltrative pulmonary abnormality asymmetrically distributed because of severe chronic pulmonary fibrosis has not changed appreciably over the past two weeks, except for the accumulation of the small left pleural effusion. moderate cardiomegaly is longstanding. et tube, left pic line, and right internal jugular line are unchanged in standard positions respectively. no pneumothorax. nasogastric tube ends in the region of the pylorus.
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comparison to. no relevant change. the right picc line has been removed. normal size of the cardiac silhouette. normal hilar and mediastinal contours. no pleural effusions. no pneumonia, no pulmonary edema.
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moderate pulmonary edema is stable. there are low lung volumes. cardiomediastinal silhouette is unchanged. lines and tubes are in standard unchanged position. there is no evident pneumothorax or enlarging pleural effusions.
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no acute intrathoracic process.
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low lung volumes causing bronchovascular crowding. bibasilar opacities most likely represent atelectasis. rightward tracheal deviation by an enlarged left thyroid lobe.
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no pneumothorax, pleural effusions or consolidations. postoperative changes from median sternotomy. bochdalek hernia is incidentally noted.
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comparison to. the right central venous access line was removed. otherwise stable chest radiograph. normal lung volumes. normal size of the heart. no pneumonia, no pulmonary edema, no pleural effusions. the elongation of the descending aorta is stable.
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mild pulmonary vascular congestion without overt pulmonary edema. no pleural effusion. possible right-sided pleural plaques with associated scarring/atelectasis.
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doppler off tube is in the distal body of the stomach.
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no definite focal consolidation. moderate pulmonary vascular engorgement.
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no acute cardiopulmonary process.
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compared to chest radiographs and. new heterogeneous pulmonary abnormalities are very asymmetrically distributed. the perihilar left upper lobe component contains vascular congestion and increased background density, looking much like pulmonary edema. the right lower lobe component is more densely opacified shallow, shows some volume loss, and is accompanied by small right pleural effusion. i cannot ascribed this simply day asymmetric pulmonary edema. instead i suggest consideration of edema in the setting of large pulmonary emboli or, alternatively, large scale aspiration. heart is slightly larger but normal size. incidental note is made of continues severe splenomegaly or other left upper quadrant mass effect. recommendation(s): repeat conventional chest radiograph, consider ct scanning if the radiographic findings are still confounding.
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no acute cardiopulmonary process. left picc ends in the distal svc.
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emphysema without superimposed pneumonia.
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no acute intrathoracic process.
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left-sided pleural effusion with underlying atelectasis and possible underlying consolidation. clinical correlation suggested. probable right basilar atelectasis.
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new moderate-sized right pneumothorax.
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since the prior study there has been interval development of bilateral, left more than right opacities in the lung bases, concerning for aspiration. there is also mild interval vascular engorgement development that might potentially reflect mild pulmonary edema. no interval increase in pleural effusion or pneumothorax is seen. cardiomediastinal silhouette is unchanged.
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mild bibasilar atelectasis. no focal consolidations concerning for pneumonia identified.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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no evidence of acute cardiopulmonary abnormalities.
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comparison to. no relevant change. normal lung volumes. no pneumonia, no pulmonary edema, no pleural effusions. normal size of the cardiac silhouette.
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no acute cardiopulmonary process. although no acute fracture or other chest wall lesion is seen, conventional chest radiographs are not sufficient for detection or characterization of most such abnormalities. if the demonstration of trauma to the chest wall is clinically warranted, the location of any referrable focal findings should be clearly marked and imaged with either bone detail radiographs or chest ct scanning.
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status post removal of left-sided chest tube with a tiny left apical pneumothorax measuring <num> mm. mild interval worsening of left basilar atelectasis. no pleural effusion. rest of the findings are stable. recommendation(s): continued followup chest radiographs to assess stability of the tiny left apical pneumothorax recommended.
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no acute cardiopulmonary process.
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et tube in standard placement. nasogastric drainage tube ends in the upper stomach would need to be advanced <num> cm to move all the side ports beyond the gastroesophageal junction. right, basal pigtail pleural drainage catheter still in place. right pleural effusion small if any. right lower lobe is densely consolidated, probably collapsed. earlier interstitial edema has resolved. multiple lung lesions, more numerous on , are resolving. heart size is normal. no pneumothorax.