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ap chest compared to : bilateral perihilar pulmonary consolidation is slightly worse today than on , and still more pronounced on the left than the right. nevertheless, the simplest explanation is that this is perihilar distribution of worsening pulmonary edema, rather than invoking a second diagnosis such as pneumonia or hemorrhage. heart is top normal size. mild mediastinal venous engorgement unchanged. et tube in standard placement. upper enteric drainage tube ends in the stomach. no appreciable pleural effusion and no pneumothorax.
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left apical pneumothorax appears to be similar to previous examination, still at least moderate. the patient is in pulmonary edema that has progressed. pigtail catheter is in place. bilateral pleural effusions are large. small right apical pneumothorax is noted, decreased in size as compared to previous examination.
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large right pleural effusion and adjacent right basal atelectasis is not significantly changed from the prior study. superimposed infection cannot be excluded. new, subtle left retrocardiac opacity is likely representative of atelectasis.
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moderately severe pulmonary edema and mediastinal venous engorgement are new. severe cardiomegaly is worsened. et tube, right pic line, nasogastric drainage tube in standard placements. pleural effusions are presumed but not large. no pneumothorax. more severe consolidation in the left lower lobe is probably acute atelectasis.
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moderately well inflated lungs with unchanged bibasilar atelectasis versus consolidation and a small left pleural effusion. interval resolution of right sided pneumothorax with unchanged position of the <num> right chest tubes. new right central venous catheter terminates at the cavoatrial junction.
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similar appearance of the right juxta hilar mass with moderate right-sided pleural effusion, loculated at the apex. otherwise no evidence of pneumonia.
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no acute intrathoracic process.
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in comparison with the study of , the dense streak of atelectasis at the left base has cleared. nevertheless, retrocardiac opacification is again consistent with atelectasis in the left lower lung. less prominent changes are seen at the right base. the left chest tube remains in place and there is no evidence of pneumothorax. the endotracheal tube remains in good position, as does the nasogastric tube. the left subclavian catheter extends to the cavoatrial junction or possibly upper portion of the right atrium. the malpositioned right subclavian catheter has been removed.
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findings suggesting mild vascular congestion. right hilar/infrahilar prominence which could potentially represent early infection, alternative considerations include vascular confluence, adenopathy, or underlying mass lesion. short-term followup imaging is recommended following appropriate treatment to exclude above-mentioned entities.
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no previous images. the cardiac silhouette is within normal limits and there is no evidence of vascular congestion, pleural effusion, or acute focal pneumonia. mild fibrotic changes are suggested in the apices with possible granuloma in the right mid lung zone. this is consistent with old healed tuberculous disease, but there is no evidence of acute reactivation.
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no convincing evidence of acute cardiopulmonary process.
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since , worsening right moderate pleural effusion. new right lower lobe atelectasis or pneumonia. stable mild cardiomegaly.
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no acute cardiopulmonary process.
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no evidence for acute cardiopulmonary process.
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no evidence of free air beneath the diaphragms.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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in comparison with the study of , there is little change. continued opacification at the left base consistent with volume loss in the lower lobes and small pleural effusion. no evidence of vascular congestion or acute focal pneumonia.
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small bilateral pleural effusions.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no pneumonia. right mediastinal contour abnormality. if there is no prior imaging already explaining this, recommend further evaluation with chest ct. findings and recommendations discussed with dr (ed) at <num>am.
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in comparison with the study of earlier in this date, there is no evidence of pneumothorax or appreciable subcutaneous emphysema. otherwise little change.
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no acute intrathoracic process.
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endotracheal tube in a somewhat high lying position, approximately <num> cm above the carina. if clinically indicated, the tube could be advanced by approximately <num> cm. right basilar opacification with volume loss including suspicion for a pleural effusion.
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no evidence of acute cardiopulmonary disease.
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focal opacity in lower lobes could reflect an infectious process in the appropriate clinical setting. mediastinal widening, likely related to central lymphadenopathy. recommend followup with ct scan. findings were communicated with by dr telephone at the time of observation at on.
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no acute cardiopulmonary process.
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small to moderate right pleural effusion with possible superimposed pulmonary vascular congestion.
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compared to chest radiographs through. multifocal pneumonia has not improved in areas of previous involvement and is more pronounced in the left midlung. heart size normal. pulmonary vasculature is probably engorged but there is no appreciable pulmonary edema or detectable pleural effusion.
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no acute intrathoracic process.
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no acute findings.
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mild vascular enlargement, left hilus, should be evaluated in clinical context to consider acute pulmonary embolus, as discussed with dr reported a normal d-dimer level, excluding pulmonary embolus.
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support lines and tubes are unchanged in position. heart size is enlarged but unchanged. there has been worsening of the opacities at the lung bases, right worse than left. there remains mild prominence of the pulmonary interstitial markings suggestive of mild fluid overload, stable. no pneumothoraces are seen.
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suspected component of interstitial edema superimposed on chronic interstitial process. cardiomegaly which has progressed since prior although some of this may be positional.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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there has been improvement of the pulmonary interstitial edema since the prior study. there is unchanged cardiomegaly. there are small bilateral pleural effusions.
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persistent low lung volumes and bibasilar atelectasis.
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continued obscuration of the left heart border favors left lower lung atelectatic changes over pneumonia. otherwise, stable chest x-ray.
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no active disease.
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normal chest radiograph.
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pulmonary vascular congestion. persistent enlargement of the cardiac silhouette.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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findings concerning for early left lower lobe pneumonia.
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no acute cardiopulmonary abnormalities
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no evidence of vascular congestion or pulmonary edema. a right base opacity, increased compared to , likely corresponds to the small to moderate pleural effusion seen on ct from the day prior. however superimposed pneumonia would be difficult to exclude. follow up to resolution is recommended.
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interval resolution of the right lower lobe density. unchanged enlargement of the cardiac silhouette.
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no acute intrathoracic process. picc line intervally removed. no foreign body seen.
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<num> right chest tubes are in similar locations. no interval increase in pleural effusion or development of pneumothorax demonstrated. left mid lung appears to be better aerated. cardiomediastinal silhouette is unchanged
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known right hilar mass is stable since prior exam on with interval increased in mild adjacent atelectasis. there is no new focal consolidation.
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bibasilar opacities are developed, may represent atelectasis, consider pneumonitis or aspiration, particularly on the right. small right pleural effusion, has worsened. no pneumomediastinum.
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normal chest radiograph. no pleural effusion, pneumothorax, or evidence of pneumonia.
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subtle opacities in lower lungs in the setting of low lung volumes likely reflect bronchovascular crowding and atelectasis. top normal heart size.
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anterior inferior left shoulder dislocation. no acute intrathoracic process. please refer to same-day ct chest for further details.
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minimal increase in right basilar opacity worrisome for infection or possible aspiration.
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pa and lateral chest compared to through : moderate cardiomegaly and mild pulmonary vascular congestion are chronic, but there is no pulmonary edema or pleural effusion. peribronchial opacification in the right lower lobe is more likely atelectasis than infection. thoracic aorta is very tortuous, but not focally dilated, and unchanged.
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endotracheal tube and right internal jugular central line are unchanged in position. a nasogastric tube is seen coursing to the level of the distal esophagus, but the tip cannot be identified, but is felt to likely be below the diaphragm. there are layering bilateral effusions. the heart remains stably enlarged, most likely reflecting cardiomegaly, although pericardial effusion should also be considered. there is stable mild pulmonary edema. no pneumothorax is seen.
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no free air below the right hemidiaphragm. right basilar atelectasis with small right pleural effusion and right hilar mass and left upper lobe mass again noted. please refer to subsequent ct of the torso for further details.
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no acute cardiopulmonary process.
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left lower lobe atelectasis and a small posterior pleural effusion. if hypoxemia persists, pulmonary embolism should be considered as a possible etiology.
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findings suggest mild pulmonary edema. suspected small pleural effusions and probable associated posterior atelectasis at the lung bases. although no nodular opacity is persistently visualized in the left mid lung on this study, this does not negate the possibility of a pulmonary nodule as mentioned in the prior report.
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no acute intrathoracic process.
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improved ventilation of the left lung bases with minimal improvement of the vascular congestion. unchanged right base atelectasis.
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stable cardiomegaly, likely mild interstitial edema. difficult to exclude an atypical infection
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as compared to radiograph, multifocal areas of consolidation involving the right lung to a greater degree than the left demonstrate interval worsening in the right lung, were these acute abnormalities are superimposed on chronic regions of scarring and emphysema. these findings have been more fully assessed by interval ct of , and are most consistent with multifocal pneumonia. exam is otherwise remarkable for proximal position of endotracheal tube, terminating <num> cm above the carina. this could be advanced by approximately <num> cm for standard positioning.
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no acute cardiac or pulmonary process. no definite rib fracture, although this study is not technically adequate to exclude a non-displaced anterior rib fracture. additionally, the position of the skin marker indicates the patient's pain is near the costochrondral junction, a site which is difficult to evaluate with conventional radiography.
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endotracheal tube tip approximately <num> cm above the carina. incomplete imaging of the left lung. near complete opacification of the right lung field.
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possible apparent mild pulmonary vascular congestion, cardiomegaly.
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as compared to the previous radiograph, the right internal jugular vein catheter has been removed. moderate cardiomegaly. no larger pleural effusions. low lung volumes. no fluid overload. mild retrocardiac atelectasis.
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left picc line tip is at the level of mid to lower svc. right basal consolidation appears to be similar to previous examination. small left basal opacity is unchanged. upper lungs are clear. there is no evidence of pneumothorax. no appreciable pleural effusion is demonstrated on the left and small amount of pleural fluid is noted on the right
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ap chest at and compared to for: tip of the right picc line is at a level <num> cm inferior to the carina, and would need to be withdrawn <num> cm to place it in the low svc. lungs clear and heart size normal. no pleural abnormality. esophagus is mildly distended with air to the level of the carina. cardiomediastinal and hilar silhouettes are otherwise unremarkable
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compared to prior chest radiographs through. substantial residual consolidation in the right lower lung has not improved. moderate cardiomegaly stable. lungs elsewhere grossly clear. no appreciable pleural effusion. no pneumothorax. cardiopulmonary support devices are in standard placements.
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no acute cardiopulmonary abnormality.
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no residual right-sided pneumothorax. suspected atelectasis of the right lower lobe.
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stable enlargement of the cardiomediastinal silhouette. again seen large hiatal hernia. possible minimal pulmonary vascular congestion.
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bibasilar opacities are likely atelectasis due to lower lung volumes noting that infection cannot be excluded.
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in comparison with the study of , there again are extensive pleural and airspace opacities on the right. opacification in the right suprahilar region is somewhat increasing, though this could reflect some obliquity of the patient. pulmonary vascular congestion continues. otherwise little change.
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no acute cardiopulmonary process.
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ap chest presented for review at <num> on : sharply marginated nearly round <num> x <num> cm high attenuation structure in the midline is probably an external artifact, such as a suction devise, but i would need orthogonal views to confirm that, as well as appropriate clinical correlation. left lower lobe consolidation, and mild leftward mediastinal shift, unchanged since , are presumably atelectasis, accompanied by increased small left pleural effusion. there is no mediastinal widening. postoperative mediastinal silhouette is unremarkable. right pleural and upper mediastinal drains are noted. no pneumothorax or right pleural effusion is present. right subclavian line ends low in the svc.
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new interstitial abnormality, more likely infection or drug reaction than cardiogenic edema. probable local recurrence, radiated right upper lobe lung cancer.
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interval placement of an endotracheal tube, which has its tip <num> cm above the carina. the nasogastric tube is seen coursing below the diaphragm with the tip not identified. a right internal jugular central line has its tip in the right atrium, unchanged. there is interval decrease in lung volumes with bibasilar airspace opacities and likely pleural effusions. these findings favor partial lower lobe atelectasis, although bibasilar pneumonia or aspiration can not be excluded. no evidence of pulmonary edema. no pneumothorax. there is widening of the mediastinal contours, which may related to vascular crowding given the markedly low volumes. followup imaging once the patient is clinically stable would be advised. cardiac size is stable and within the upper limits of normal given portable technique.
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appropriate position of ng tube and et tube. opacity at right base may be aspiration or pneumonia.
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new fan-shaped opacity in left mid lung may represent atelectasis however consider following for possible pneumonia. stable residual hemorrhage and atelectasis in the left upper and lower lobes. no hemothorax or pneumothorax.
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compare to prior chest radiographs. previous mild pulmonary edema has improved, pulmonary and mediastinal vascular engorgement and moderate cardiomegaly are unchanged. pleural effusion is small if any. no pneumothorax. et tube and esophageal drainage tube in standard placements.
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no definite acute intrathoracic process.
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ap chest compared to : severe multifocal pulmonary consolidation, hemorrhage, and/or pneumonia have not improved. mild interstitial edema has developed in the uninvolved portions of the lungs. heart size is top normal. no pneumothorax. pleural effusions are small, if any. right jugular line, et tube, and newly placed upper enteric drainage tube are in standard placements.
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in comparison to previous radiograph of <num> day earlier, endotracheal tube has been slightly advanced, now terminating approximately <num> cm above the carina. this could be withdrawn a few cm for standard positioning. note is also made of subcutaneous emphysema in the right supraclavicular region with apparent adjacent surgical packing material, not well evaluated on this portable chest exam. within the chest, bilateral asymmetrically distributed airspace opacities have worsened in the left mid lung and slightly improved in the right lower lung. these findings could be due to multifocal aspiration and/or infectious pneumonia.
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no acute cardiopulmonary process
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in comparison with the study , following cardiac surgery the tip of the endotracheal tube lies approximately <num> cm above the carina. nasogastric tube extends well into the stomach. left chest tube is in place and there is no definite pneumothorax. opacification at the left base is consistent with volume loss in the lower lobe probable small effusion. there is suggestion of some opacification in the right upper zone, which could represent aspiration in the appropriate clinical setting.
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in comparison with the study of , there again is patchy opacification at the left base. this could represent merely atelectasis, though in the appropriate clinical setting superimposed pneumonia would have to be considered. otherwise little change.
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no acute cardiopulmonary process.
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no focal consolidation. small bilateral pleural effusions. numerous known bilateral pulmonary metastases are better evaluated on previous chest ct.
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no radiographic evidence for pneumonia. unchanged left mediastinal masses and left lower lobe nodule compatible with metastases.
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no evidence of pneumonia or congestive heart failure. findings suggestive of copd.
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acute left through <num>th rib fractures. no pneumothorax. mild interstitial edema andleft basilar opacity which could represent any combination of atelectasis or consolidation.
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as compared to the previous radiograph, the patient has received a right internal jugular vein catheter. the tip of the catheter projects over the cavoatrial junction. there is no evidence of complications, notably no pneumothorax. the lung volumes are low. signs of mild fluid overload are present. no larger pleural effusions. no pneumothorax. no pneumonia. minimal retrocardiac atelectasis.
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in comparison with the study of from an outside facility, there has been substantial decrease in the bilateral pulmonary opacifications. monitoring and support devices have been removed. the cardiac silhouette is within normal limits. mild prominence of the ascending and descending aorta raise the possibility of underlying hypertension. some indistinctness of pulmonary vessels could reflect elevated pulmonary venous pressure. are mild focal areas of opacification suggested at the bases. in view of the relatively low lung volumes, these could merely reflect areas of atelectasis. however, in the appropriate clinical setting, superimposed pneumonia could be considered.
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no acute cardiopulmonary process.
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