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no evidence of acute disease.
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no acute intrathoracic process.
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no acute intrathoracic abnormality.
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volume loss versus small infiltrate only seen on the lateral film
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no significant change compared to with chronic elevated hemidiaphragm/extrapleural fat in the right costophrenic angle. no acute cardiopulmonary process.
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no appreciable change since , right lower lobe is probably still collapsed, moderate right pleural effusion has increased since. left lung is clear aside from retained barium in the airway. heart size normal. the chest cta showed extensive central adenopathy and severe narrowing of the right lower lobe bronchus concerning for widespread malignancy.
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no acute cardiopulmonary process.
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persistent left basilar opacity concerning for pneumonia and small left pleural effusion. new right basilar opacity could reflect pneumonia or aspiration.
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no acute cardiopulmonary process. mild cardiomegaly.
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no acute cardiothoracic process.
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retrocardiac opacity, which may represent atelectasis but cannot exclude pneumonia respiration the right clinical setting.
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compared to chest radiographs at. new left apical pleural drain in place. new left lower lobe atelectasis. left pneumothorax minimal if any. subcutaneous emphysema left lower thoracoabdominal wall reflects rib resection, presumably entailed with spinal surgery. heart size normal. right lung clear aside from mild basal atelectasis. no pulmonary edema. an esophageal drainage tube ends in the distal stomach. tip of the endotracheal tube is at the upper margin of the clavicles, no less than <num> cm from the carina, <num> or <num> cm above standard placement.
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lung volumes are lower than before which may account for region of greater opacification at the right lung base medially. lateral view was performed at even a lower level of inspiration. if symptoms persist, i would recommend repeat chest radiographs at full inspiration incorporating oblique views to improve the evaluation of the lower lobes. upper lungs are clear. the cardiomediastinal and hilar silhouettes are normal and there is no pleural abnormality.
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interval decrease in extent of right upper lobe opacity, but was some residual remaining.
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no acute cardiopulmonary process. no free air below the diaphragm.
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findings compatible with pericardial effusion, likely related to pericarditis as per clinical history. findings reported to dr by phone at <num> on.
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no evidence of acute cardiopulmonary disease.
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no evidence of acute cardiopulmonary disease.
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status post fiducial placement at the level of the left hilus, clips projecting over the right aspect of the mediastinum. postoperative appearance of the right hilus. laterally and apical a, there is a millimetric left pneumothorax without evidence of tension. normal size of the cardiac silhouette. mild elongation of the descending aorta.
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right main stem bronchial intubation, discussed with dr at approximately on date of exam. retraction by <num> cm is advised. perihilar air bronchograms likely indicative of consolidation in this region.
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low lung volumes. no confluent consolidation.
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as compared to the previous radiograph, the tip of an endotracheal tube is seen at the upper aspect of the image. the tip projects <num> cm above the carina. the lung volumes are low. retrocardiac atelectasis. normal size of the cardiac silhouette. right picc line has been removed. no pulmonary edema. no larger pleural effusions.
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normal postoperative appearance following right upper lobectomy. no pneumothorax or pleural effusion, pleural drainage catheter in place. mild atelectasis, left midlung. lungs otherwise clear. normal cardiomediastinal and hilar contours.
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no acute findings.
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new left lower lobe opacification concerning for developing pneumonia.
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in comparison with the study of , there is little interval change. the cardiac silhouette is at the upper limits of normal in size and there is no evidence of acute pneumonia, vascular congestion, or pleural effusion. streak of atelectasis is seen at the left base.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no pneumothorax status post removal of chest tube. no significant interval change in appearance of left base with persistent atelectasis and small pleural effusion.
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severe subcutaneous emphysema in the right chest and both sides of the neck has probably not worsened since after increasing substantially relative to. small right hydro pneumothorax collected laterally is unchanged, pleural drain still in the right chest. pulmonary vascular congestion has increased since. heart size is normal. epidural infusion catheter is coiled in the midline.
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no acute cardiopulmonary abnormality. soft tissue prominence of the right hilum corresponds to enlarged lymph node on the same-day cta.
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mild pulmonary edema has worsened. cardiomegaly is stable. there is no pneumothorax. there are probably bilateral pleural effusions, small larger on the right side
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unchanged cardiomegaly and retrocardiac atelectasis without acute process.
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interval development of a <num> cm elliptical opacity in the mid left upper lung. the incomplete sharp borders suggest an extra pulmonary process located in either the pleura or chest wall. ct is recommended for further evaluation to exclude a malignant lesion. interval progression of loss of height of a mid thoracic compression fracture.
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status post right chest tube placement with near resolution of previous large right hydro
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left mid lung atelectasis/ scarring. no focal consolidation to suggest pneumonia.
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vague ill-defined densities in the left mid zone (lignula), ? left lower zone laterally, and possibly also in the right mid zone, which could represent early infectious infiltrates in an immunocompromised patient. in retrospect, faint opacities were likely present in these areas on the radiographs from. dr was paged at the time of discovery at and findings were discussed with him shortly thereafter (, phone).
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no acute cardiopulmonary process. no evidence of pneumothorax. if clinical concern for spine injury, suggest dedicated imaging of that region of the spine.
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findings compatible with acute pulmonary edema with possible layering pleural effusions. superimposed infectious/inflammatory process cannot be excluded. significant left apical density may represent a loculated pleural effusion versus a mass. followup of this will be necessary after treatment.
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no subcutaneous air is identified.
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ett appropriate in position. no definite acute cardiopulmonary process.
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no acute intrathoracic process.
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new, lateral, mid right lung opacity concerning for pneumonia. unchanged, dense, retrocardiac consolidation with associated moderate pleural effusion is concerning for pneumonia and atelectasis. improved pulmonary edema.
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enlarged heart, pulmonary edema and left lower lobe pleural effusion, present on prior examinaion and unchanged, most consistent with mild congestive heart failure.
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normal chest radiograph.
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no acute cardiopulmonary process.
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suspect pronounced background copd, with overall parenchymal scarring and also biapical pleural and parenchymal scarring, detailed above. mild cardiomegaly. no chf or focal infectious infiltrate identified. <num>-mm nodule in right upper zone. further evaluation with chest ct scan would be indicated when the patient is stable.
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in comparison with the study of , there is little change in the diffuse bilateral pulmonary opacifications consistent with significant aspiration pneumonia. mild indistinctness of pulmonary vessels could reflect some degree of over-hydration. monitoring support devices are essentially unchanged.
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findings most consistent with moderate interstitial pulmonary edema, accompanied by pleural effusions. attenation in follow-up suggested regarding more confluent opacity at the right lung base although edema is again the suspected etiology; coinciding pneumonia is not excluded, however.
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continued clearing, right lung wedge resection site. no pneumothorax or pleural effusion. extensive pulmonary fibrosis has progressed over the past years. heart size top-normal.
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no acute cardiopulmonary process. the paucity of upper lung markings at the bilateral apices suggests emphysema.
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unchanged retrocardiac opacification since , with no definite evidence of pneumonia.
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large right pleural effusion which developed between and has redistributed, but probably not changed overall in volume since. heart is normal size. bulging left posterior mediastinal contour just above the diaphragm is probably due to displacement by the right pleural effusion. aside from relaxation atelectasis in the right lower lobe, right upper lobe and left lung are clear. no pneumothorax.
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chronic fibrotic changes in the lungs, not significantly changed compared to prior chest x-ray. no definite superimposed consolidation.
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interval placement of a right internal jugular catheter which terminates in the low svc. otherwise, no significant change from <num> hour earlier.
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increasing pulmonary congestion with development of bilateral pleural effusion most likely related to chf. non-distended azygos vein speaks against general fluid overload and favors assumption of left-sided chf.
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no acute cardiopulmonary process.
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since a recent radiograph from earlier today, a feeding tube has been placed with tip terminating in the proximal stomach. pre-existing nasogastric tube extends to the region of the gastroduodenal junction. the chest is incompletely imaged on this radiograph targeted for feeding tube assessment but the thorax has been more fully assessed by a a recent chest radiograph from several hr earlier.
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appropriate position of all lines and tubes. resolution of pulmonary edema.
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a right basilar chest tube remains in place and there continues to be bilateral blunting of the costophrenic angles posteriorly consistent with small effusions. a focal lucency surrounding the tip of the pleural catheter is unchanged and could represent a tiny loculated hydro pneumothorax. no large pneumothorax is seen. there is stable scarring within the right lower lung and streaky opacity at the left base likely reflecting atelectasis or scarring. no focal airspace consolidation is seen to suggest pneumonia. no pulmonary edema. degenerative changes in the thoracic spine. overall, there is no significant interval change since.
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moderate left-sided pleural effusion. widespread airspace disease in the right lower lung worrisome for pneumonia. cholelithiasis.
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no acute cardiopulmonary process.
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as compared to the previous examination, the nasogastric tube is now foley image. the tip projects over the middle parts of the stomach. there is no evidence of complications, notably no pneumothorax. otherwise the radiograph is unchanged, with the exception of a right internal jugular vein catheter that has been removed in the interval.
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bilateral pleural effusions with bibasilar atelectasis increased compared to radiograph from a day prior. postoperative appearance shows more narrowing of lower trachea with improved upper trachea compared to most recent radiograph.
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no acute cardiopulmonary abnormality.
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patchy and linear mid and lower lung opacities favoring atelectasis. followup radiographs may be helpful to exclude focal pneumonia in left lower lobe.
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improved pulmonary vascular congestion from. improved left basilar atelectasis. unchanged small bilateral pleural effusions.
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in comparison with the study of , there has been clearing of the small right apical hydro pneumothorax. the right supraclavicular subcutaneous gas has cleared. mild right basilar atelectatic changes again seen. multiple healing or healed rib fractures are again seen on the left.
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there is unchanged cardiomegaly. there is again seen diffuse prominence of the pulmonary interstitial markings bilaterally. this may be seen in the setting of infection or pulmonary edema, unchanged. there is atelectasis at the lung bases, also stable. there are no pneumothoraces.
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no evidence of acute disease.
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no evidence of acute cardiopulmonary process.
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as compared to , the pre-existing parenchymal opacities are improved, notably in the left lung and in the right lung apex. an opacity in the left lung bases and the right lower lung zones is not substantially changed. massive intestinal distension is constant in severity.
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no acute intrathoracic process. vp shunt noted.
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no acute cardiopulmonary process. nodular opacity over the left lung apex. this could potentially be within the bone or in the parenchyma from pulmonary nodule or scarring.
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improved aeration of the right lung since the radiograph. postoperative changes throughout the left lung are stable.
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probable chronic lung disease related to asbestos exposure no acute cardiopulmonary process
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ap chest compared to : previous pulmonary edema has cleared, right lower lobe consolidation has improved, but left lower lobe consolidation is still very large, very dense, and accompanied by a moderate-sized left pleural effusion. of note, there has been little change in the radiographic findings since when a concurrent chest ct showed no pericardial effusion, and left lower lobe collapse, attributable to the large minimally hemorrhagic left pleural effusion. no pneumothorax.
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small right apical pneumothorax is stable since the prior study. multiple right-sided rib fractures are better assessed on the prior study which included the rib series.
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MIMIC-CXR-JPG/2.0.0/files/p18902452/s55851190/7471e739-f2246c43-54684b79-3b9e7fa5-7f2e05fd.jpg
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no acute cardiopulmonary abnormality. no subdiaphragmatic free air.
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MIMIC-CXR-JPG/2.0.0/files/p16248139/s57586934/8e47a05e-2b662d29-bb35e575-11c5e27d-e4a73d1a.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p15588817/s58286115/5f2309b2-a6083ce7-f2f0cf54-4f639ac4-fd80b276.jpg
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although there are no recent prior chest radiographs, the study is read in conjunction with the chest cta performed on showing extensive pulmonary emboli, large left hilar mass infiltrating mediastinum, left pleural effusion, lung nodules and probable pulmonary infarcts, all visible on this chest radiograph. today's chest radiograph shows that the heart is normal size and the right lung shows no pulmonary edema. the left lung is partially obscured by pleural effusion and probable worsening left lower lobe atelectasis. mild edema in the upper lobe is a function of lymphovenous congestion by the left hilar mass. no pneumothorax.
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MIMIC-CXR-JPG/2.0.0/files/p17195386/s54750022/eed3316d-d0cdb23a-8f63ef28-df88d4a2-a15840c6.jpg
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persistent unchanged bilateral pleural effusions and bibasilar atelectasis. interval improvement in pulmonary edema.
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MIMIC-CXR-JPG/2.0.0/files/p15566609/s54869378/e2eee053-f658b831-de3cdd09-94940aac-88fb1030.jpg
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large retrocardiac atelectasis have markedly increased. cardiomediastinal contours are unchanged. small bilateral effusions larger on the right are grossly unchanged. vascular congestion is stable. right chest tube remains in place. left subclavian catheter tip is in cavoatrial junction. mediastinal drain is unchanged
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MIMIC-CXR-JPG/2.0.0/files/p13923862/s53895781/f8f84793-99b5b55e-0b480a3d-acd1e81c-4260b6e9.jpg
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no acute cardiopulmonary abnormalities
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MIMIC-CXR-JPG/2.0.0/files/p19133405/s55781332/565a8494-6c39f53c-809345cc-6e53090f-56f621dc.jpg
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no acute findings in the chest.
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MIMIC-CXR-JPG/2.0.0/files/p15911529/s50329542/41811dc3-c03a8c6d-a316dd7f-5733949b-00331055.jpg
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interval enlargement of the right pleural effusion and pulmonary vascular congestion. please note that underlying infection at the right lung base cannot be excluded.
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MIMIC-CXR-JPG/2.0.0/files/p13063188/s58185963/121a8e80-3868f6e0-c44ac38e-ed1da7f0-3e00d490.jpg
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cardiomegaly and enlarged pulmonary artery suggesting pulmonary hypertension. no superimposed acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p16169853/s52252325/50d09d11-965e0ebb-6206b056-d456b2c9-ce58897d.jpg
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status post endotracheal intubation. orogastric tube terminates in the stomach, although the side hole marker is located above the gastroesophageal junction. case discussed with dr at pm on.
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MIMIC-CXR-JPG/2.0.0/files/p14825395/s53800537/4dc89d5a-f8e9f290-96be559b-9d7c2321-e1767bd4.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p18297386/s53229242/ef1a6a89-252d2dc5-4be0661a-aa8492b9-782594ac.jpg
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in comparison with the study of , there is little change. right chest tube remains in place and there is no pneumothorax. diffuse bilateral pulmonary opacifications are again seen.
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MIMIC-CXR-JPG/2.0.0/files/p17414442/s52091168/26052119-7ada8bd3-43356b31-7b9348fd-6ede490a.jpg
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cardiac findings suggestive of systemic hypertension but no evidence of significant pulmonary vascular congestion. general appearance of some emphysema and moderate widening of superior mediastinum suggestive of intrathoracic thyroid.
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MIMIC-CXR-JPG/2.0.0/files/p10921047/s57457390/bdee5f89-ea9e74e1-a26c7716-2046d54b-369f2d80.jpg
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no evidence of acute cardiopulmonary disease.
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MIMIC-CXR-JPG/2.0.0/files/p12500505/s57422397/4843815d-b4a59979-cdd6e3ca-ae3f0165-916abada.jpg
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no definite acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p14495401/s52094857/34c44071-ad3880c9-d369ca7a-ee5db2ad-8ae64c11.jpg
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in comparison with the study of , the monitoring and support devices are unchanged. there is increasing opacification at the bases, especially on the right. although this could merely reflect atelectatic change, in the appropriate clinical setting superimposed pneumonia would have to be seriously considered. residual contrast material is again seen in the visualized portion of the colon.
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MIMIC-CXR-JPG/2.0.0/files/p16976843/s54180399/3cf304e8-ac08aa0f-fed5747e-3507ce67-cb6acb9e.jpg
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no evidence of active tuberculosis. possible tiny calcified granuloma versus vessel on in the right upper lung.
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MIMIC-CXR-JPG/2.0.0/files/p15879564/s54933442/53d83fe7-b33f2efd-8ed31aeb-fec308f3-45a064b4.jpg
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no acute cardiopulmonary process.
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MIMIC-CXR-JPG/2.0.0/files/p16283409/s59329775/92e44210-58457d8e-863ba0e6-752ec023-d9760c86.jpg
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no significant interval change.
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MIMIC-CXR-JPG/2.0.0/files/p13339327/s56749419/b3687d4f-a24312ef-b2cb0609-c0064308-90b88935.jpg
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no acute cardiopulmonary process. no evidence of free air beneath the diaphragms.
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MIMIC-CXR-JPG/2.0.0/files/p19027535/s55326485/4eb14255-32c85103-88f9c56f-9117adee-b8b1a3c2.jpg
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normal chest radiograph.
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MIMIC-CXR-JPG/2.0.0/files/p11669811/s51911606/7c752999-dd0d361a-c2bd6f68-434e18d5-2c23f961.jpg
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hyperinflated lungs. no radiographic evidence of interstitial lung disease. potential pulmonary hypertension, please correlate with echocardiography.
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