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small right pleural effusion may have reaccumulated since following removal of the right pleural drains. right apical pneumothorax is tiny. new consolidation in the left lower lobe could be atelectasis, raises concern for pneumonia. pulmonary vascular congestion suggests some cardiac decompensation. right jugular line ends in the mid svc.
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et tube tip is <num> cm above the carinal. ng tube tip is in the stomach. pacemaker lead terminates in the right ventricle. large bilateral pleural effusions appear to be slightly increased since the prior study. pulmonary edema is moderate, unchanged
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no pneumonia.
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right chest tube remains in place. there has been interval appearance of extensive subcutaneous emphysema and interval enlargement of a small-to-moderate sized right pneumothorax. close followup imaging is advised. the left lung remains grossly clear, although there is minimal linear opacity at the left costophrenic angle, which may represent post-inflammatory scarring or subsegmental atelectasis. single-lead left-sided pacer remains unchanged in position. overall, cardiac size is stable. results were conveyed to the patient's nurse, , in the intensive care unit on at the nurse stated that the chest tube has been placed back to suction.
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persistent right mid to lower lung consolidation, potentially atelectasis noting that infection is possible. right-sided pleural effusion persists.
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no acute cardiopulmonary process.
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right internal jugular central venous catheter has been advanced and now terminates in the lower superior vena cava, with no visible pneumothorax. allowing for artifact overlying the left thoracoabdominal junction region, overall appearance of the chest is otherwise not appreciably changed since recent study of earlier the same date.
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in comparison with the study of , there has been placement of an endotracheal tube with its tip approximately <num> cm above the carina. it could be pulled back about <num> cm for more optimal position. right ij catheter extends to the cavoatrial junction and the nasogastric tube extends well into the stomach. continued enlargement of the cardiac silhouette with pulmonary vascular congestion. hazy opacification bilaterally is consistent with layering effusions with volume loss most prominently involving the left lower lobe in the retrocardiac region.
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in comparison with the study , the monitoring and support devices are unchanged, as is the appearance of the heart and lungs. no definite evidence of acute pneumonia. however, the retrocardiac area cannot be properly evaluated in the absence of a lateral view.
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similar as on next preceding examination of , the portable examination shows unchanged findings. consider detailed chest examinations in this patient who allegedly has history of lung cancer.
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copd. right upper lobe bronchial inflammation or early pneumonia.
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no acute cardiopulmonary process.
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scattered subsegmental atelectasis, small right pleural effusion. please refer to subsequent cta chest for further details.
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there is a likely status of the right-sided lobectomy, with postoperative paramediastinal changes at the level of the lung apex and an elevation of the right hemidiaphragm. on the current image, no definite signs of recurrence or malignancy are present, but comparison to previous images is required to make certain determination. no evidence of acute lung disease such as pneumonia or pulmonary edema. no pleural effusions. normal size of the cardiac silhouette. normal hilar and mediastinal contours.
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no evidence of acute disease.
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comparison to. slightly increasing right pleural effusion. monitoring and support devices are stable. stable borderline size of the cardiac silhouette. no overt pulmonary edema. no pneumonia.
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unchanged right-sided pleural effusion with right-sided percutaneous drainage catheter and right hilar mass and nodularity. no new consolidation.
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as compared to the prior radiograph of <num> day earlier, cardiomegaly is accompanied by pulmonary vascular congestion, and worsening bibasilar consolidation and adjacent pleural effusions, now moderate in size.
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lower lung volumes causing bronchovascular crowding. however, more indistinct, cephalized pulmonary vessels suggests a component of vascular congestion. no new focal consolidation.
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no acute cardiopulmonary process.
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no acute thoracic injury.
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new bibasilar linear opacities likely secondary to atelectasis; however, an acute infectious process cannot be excluded. new small left pleural effusion.
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no significant interval change when compared to the prior study.
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no signs of pneumonia.
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moderately severe pulmonary edema has returned. there is new consolidation in the right upper lobe, and this is either recurrence of asymmetric pulmonary edema or another focus of pneumonia in addition to the persisting consolidation in the right and left lower lobes. moderate cardiomegaly unchanged. bilateral pleural effusions are small. no pneumothorax.
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new moderate right pleural effusion with at least right lower lobe segmental collapse no findings suggest cardiac decompensation.
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peribronchial thickening in the bilateral lower lungs, with ill-defined basilar opacities, may be seen in atypical pneumonia.
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no evidence of pneumonia. mild bronchial wall thickening could reflect bronchitis.
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interval worsening of moderate pulmonary edema with small-to-moderate sized bilateral pleural effusions.
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no acute intrathoracic disease.
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as compared to prior radiograph of <num> day earlier, cardiomegaly is accompanied by worsening pulmonary vascular congestion. worsening interstitial opacities in small pleural effusions are likely due to hydrostatic edema and less likely an atypical pneumonia. no definite new or worsening areas of consolidation to suggest bacterial pneumonia.
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no acute cardiopulmonary abnormality.
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no significant interval change in size of large right basilar pneumothorax. no evidence of tension. extensive subcutaneous emphysema. bibasilar opacities may represent atelectasis or aspiration.
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slight interstitial abnormality suggesting mild interstitial congestion or fluid overload.
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decreased size of small left pleural effusion. otherwise, no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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no previous images. cardiac silhouette is within normal limits and there is no vascular congestion or pleural effusion. mild hyperexpansion of the lungs could reflect underlying chronic pulmonary disease. however, no evidence of acute focal pneumonia.
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no acute cardiopulmonary process.
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mild pulmonary edema with moderate size right and small left bilateral pleural effusions. bibasilar airspace opacities could reflect compressive atelectasis. infection or aspiration at the lung bases however is not completely excluded.
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no previous images. the heart is normal in size and there is no evidence of vascular congestion, pleural effusion, or acute focal pneumonia.
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no evidence of acute disease.
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new nasogastric tube with course suggestive of malpositioning within the left lower lobe bronchus. removal or repositioning is advised. otherwise no significant interval change.
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no acute intrathoracic process.
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esophageal drainage tube ends in the upper stomach. et tube and left subclavian line are in standard placements. moderate left pleural effusion and small right pleural effusion have increased. heart size is normal. previously questioned atelectasis early consolidation in the superior segment of the left lower lobe is less apparent, and may have improved. heart size normal. no pneumothorax.
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post sternotomy wires are stable. heart size and mediastinum are unchanged including extensive tortuosity of the descending aorta, the seen back on. bibasal linear atelectasis are re- demonstrated, potentially representing scarring. lobe lesion of the diaphragm is noted bilaterally. there is also evidence of hyperinflation and coronary stents. there is no pleural effusion or pneumothorax appreciated as well is no new consolidations seen. compression fractures better appreciated on the lateral view appear to be progressed as compared to.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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question small amount of free air. recommend clinical correlation and followup when the patient is able.
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lucency at the left lung apex without definite pneumothorax. if clinically concerned, repeat radiograph can be performed in a less lordotic position.
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no acute pulmonary process identified. platelike opacities in left lung, similar to the prior film. no new opacity identified.
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et tube tip is <num> cm above the carina and should be pulled back at least <num> cm. heart size is mildly enlarged. mediastinum is unchanged. lung volumes are low with bibasal atelectasis most likely related to low lung volumes.
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low lung volumes, but otherwise no acute process.
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in comparison with the study of , the heart is normal in size and is no vascular congestion, pleural effusion, or acute focal pneumonia.
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in comparison with the study of , there has been some decrease in the still substantial atelectatic changes at the right base. the the retrocardiac atelectasis is less prominent than on the previous study. and has eminence changes in the right upper lung are unchanged.
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interval increase in right lung opacification likely from atelectasis rather than pneumonia. worsening bilateral pleural effusion and persistent left lower lobe atelectasis. monitoring and support devices are all unchanged.
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no signs for acute cardiopulmonary process.
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no acute cardiopulmonary process.
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endotracheal tube terminates <num> cm above the carina. enteric catheter terminates in expected location of the body of stomach.
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low lung volumes with focal left basilar opacity which could be secondary to atelectasis although infection is not entirely excluded.
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mild pulmonary vascular congestion. bibasilar interstitial abnormality, either atelectasis or chronic changes. no evidence of superior mediastinal widening.
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no acute cardiopulmonary abnormality.
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as compared to the previous radiograph, no relevant change is seen. a subtle lingular scar, better visualized on the frontal than on the lateral radiograph, is unchanged in extent and severity. mild overinflation and blunting of the right costophrenic sinus, likely caused by a pleural thickening. the no acute changes. no pneumonia, no pulmonary edema. no larger pleural effusions.
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in comparison with the study of , this and placement of tube. the opaque tip lies just distal to the esophagogastric junction. if possible, the tube should be pushed forward several cm. the heart and lungs are not adequately seen, but show no gross change from the previous study.
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resolving right lower lobe pneumonia.
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MIMIC-CXR-JPG/2.0.0/files/p16267288/s54255077/ae47d418-932522a5-eba553fb-96b9fcb9-589ed190.jpg
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no acute cardiopulmonary abnormality.
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pulmonary edema, with right greater than left lung involvement.
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hyperinflated lungs without signs of pneumonia or chf. mild cardiomegaly.
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normal chest radiograph.
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right lower lobe opacity is worrisome for pulmonary contusion, pneumonia or aspiration in the appropriate clinical setting. endotracheal tube in appropriate position.
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no acute cardiopulmonary process. no evidence of congestive failure.
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no acute intrathoracic process.
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compared to prior chest radiographs through. <num> frontal images of the chest both exclude the right lateral chest wall. they show sequential advancement of the esophageal feeding tube from the mid esophagus to the upper stomach. lung volumes remain quite low, vascular congestion interstitial abnormality in the left lower lung and at least small left pleural effusion are unchanged. no left pneumothorax. left pic line ends in the mid svc. heart and mediastinum are enlarged but difficult to assess because of patient rotation.
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no focal consolidations concerning for pneumonia identified.
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no acute cardiopulmonary process
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no free air under the diaphragms. no acute cardiopulmonary abnormality.
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persistent, marked atelectasis, left lower lobe. unchanged displacement of osteotomy at the fourth posterior right rib with no evidence of hemorrhage or pneumothorax.
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no acute cardiopulmonary process. these findings were discussed by dr with dr telephone at on.
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no pulmonary edema, pleural effusion, or focal consolidation.
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no acute cardiopulmonary abnormality.
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as compared to the previous radiograph, there is an unchanged millimetric right apical pneumothorax. a right chest tube is in constant position. the known right rib fractures are also unchanged. moderate cardiomegaly persists. normal appearance of the left lung.
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MIMIC-CXR-JPG/2.0.0/files/p13044775/s51590556/b8815435-f5fbf4b8-c4b3d0cb-ea0ad17b-63c05cb6.jpg
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no acute cardiopulmonary process.
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streaky bibasilar opacities compatible with atelectasis. unchanged tubular opacity in the right upper lobe, previously characterized on ct as an area of mucous plugging.
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normal chest radiograph.
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compare to prior chest radiographs through :<num>. bibasilar consolidation has worsened since , stable since. small pleural effusions are presumed. heart size normal. lines and tubes in standard placements.
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retrocardiac opacity, likely due to atelectasis but possibly due to pneumonia in the appropriate setting.
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compared to chest radiographs since , most recently and. it is difficult to compare <num> g bedside radiographs with earlier conventional chest radiographs. the apparent increase in heart size may not be real, and the suggestion of new alveolar opacification may be a function of decreased resolution. possibility of biventricular heart failure is raised however. this does not look like pneumonia. the pulmonary changes are not prominent and therefore probably not accelerated pulmonary fibrosis.
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no acute cardiopulmonary process. large gastric air-fluid level.
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in comparison with the study of , the right chest tube has been removed. there is again a small apical pneumothorax on this side. opacification at the right base is consistent with a combination of pleural effusion and volume loss. less prominent effusion and atelectasis is seen on the left.
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streaky bibasilar opacities potentially atelectasis. cannot entirely exclude pneumonia.
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findings consistent with pneumonia in the right lower lobe. discussed with dr on , by telephone.
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ap and lateral chest compared to. there is persistence of the region of mild peribronchial opacification in the right lower lung seen laterally on the frontal view, not clearly localized on the lateral. it is not abnormal enough to characterize as pneumonia, but if there is high clinical suspicion of pneumonia, an additional radiographic confirmation is needed and oblique view should be obtained. heart is mildly enlarged, but there is no pulmonary edema, pleural effusion or vascular engorgement.
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interval resolution of bilateral pleural effusions. there is no pulmonary edema.
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findings most suggestive of mild pulmonary congestion.
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no acute cardiopulmonary abnormality. copd.
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no relevant change as compared to the previous image. the right internal jugular vein catheter is in unchanged position. low lung volumes. moderate cardiomegaly with tortuosity of the descending aorta. mild fluid overload but no overt pulmonary edema. small retrocardiac atelectasis is not substantially changed. no pleural effusions. no pneumonia.
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pa and lateral chest compared to : region of questioned right middle lobe pneumonia has not become more radiodense with the administration of contrast agent. i doubt there is pneumonia there or elsewhere in the lungs. if there is clinical justification for increasing the level of certainty in diagnosing pneumonia--, for example-- imaging of this area should start with a left anterior oblique chest radiograph, and as a second step limited ct scanning of the right middle lobe area. there is no pleural effusion or evidence of central adenopathy. heart size is normal.
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no previous images. there is enlargement of the cardiac silhouette, accentuated by a relatively low lung volumes. moderate tortuosity of the aorta. no evidence of acute pneumonia, vascular congestion, or pleural effusion. no convincing evidence of sclerotic bone metastases.
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there no prior chest radiographs available for review. heart size top-normal. lungs clear. normal mediastinal and hilar contours. no pleural abnormality.
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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease. continued enlargement of the cardiac silhouette with dual channel pacer and leads extending to the right atrium and apex of the right ventricle. no vascular congestion or pleural effusion or acute focal pneumonia.
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no radiographic evidence for acute cardiopulmonary process.
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