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stable, hyperinflation of the lungs bilaterally, consistent with known copd. basal predominant lucency is suggestive of alpha-<num> antitrypsin deficiency. no acute cardiopulmonary abnormality.
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heart size and mediastinum are stable including mild bb tortuosity of the aorta and mediastinal widening. bibasal opacities, primarily linear but with some nodularity as similar to previous study and most likely reflect chronic interstitial lung disease. no new abnormalities to suggest acute infection currently seen.
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interval resolution of left apical pneumothorax.
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compared to prior chest radiographs , most recently. no pneumonia or pulmonary edema. heart size top- normal. fullness in the right lower paratracheal station could be due to vascular engorgement or new adenopathy. no appreciable pleural abnormality. recommendation(s): conventional chest radiographs should be performed when feasible.
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no evidence of acute cardiopulmonary disease.
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small right pleural effusion, not significantly changed compared to the prior study.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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low inspiratory volumes with bibasilar atelectasis. in this setting, an infectious infiltrate would be difficult to exclude. small left and ? tiny right effusion. upper zone redistribution. given low lung volumes, doubt overt chf. small amount of free intraperitoneal air deep to the right hemidiaphragm, for which clinical correlation is requested. has there been recent iatrogenic introduction of air into the abdomen? otherwise, viscus perforation would be considered. addendum -- review of the patient's orders refers to peritoneal dialysis. in the appropriate clinical setting, this could account for the air seen beneath the diaphragm.
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no acute cardiopulmonary abnormality.
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streaky bibasilar atelectasis. no focal consolidation is identified to suggest pneumonia.
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increased interstitial markings throughout the lungs bilaterally which can be seen in the setting of atypical infection or interstitial edema, likely superimposed on underlying emphysema.
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left perihilar opacification with associated left upper lobe linear atelectasis that could represent pneumonia or a left hilar obstructive mass. recommendation(s): recommend ct chest for further evaluation.
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no evidence of acute cardiopulmonary disease. pleural-based density which should be evaluated with chest ct when clinically appropriate less prior studies are available to show long-term stability. comparison from is still pending, however. check priors
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no evidence of acute cardiopulmonary disease.
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endotracheal tube, right subclavian picc line and feeding tube are unchanged in position, although the tip of the feeding tube is not included on the study. a right brachiocephalic venous stent remains in place. the patient is status post median sternotomy with valve replacement and the cardiac and mediastinal contours are unchanged. there has been interval improvement in appearance of the interstitium, suggesting a resolving component of edema. there is retrocardiac opacity and more focal patchy opacity at the right medial lung base, both of which could reflect areas of atelectasis, although an infectious process should also be considered. no evidence of pneumothorax, although the sensitivity to detect a pneumothorax is diminished given semi-supine technique.
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in comparison with the study of , the pulmonary vascular congestion has substantially improved, as has the bilateral pleural effusions. cardiac silhouette remains within normal limits in size. bilateral apical pleural thickening and calcification is again seen.
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clear lungs.
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no acute cardiopulmonary process.
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#NAME?
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as compared to the previous radiograph, no relevant change is seen. status post icd placement. low lung volumes. no pneumothorax. no pleural effusion. moderate cardiomegaly. no pulmonary edema.
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new bilateral lower lobe pneumonia with a small left parapneumonic effusion.
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as compared to , cardiomediastinal contours are stable. with the exception of minor areas of linear scar atelectasis, lungs are grossly clear, and note is made of a persistent small right pleural effusion.
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no acute cardiopulmonary process.
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right middle and lower lung zone opacities concerning for aspiration pneumonitis versus pneumonia.
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cardiomegaly is severe, unchanged. pacemaker leads are unchanged. there is interval progression of vascular congestion and interstitial pulmonary edema. no pneumothorax. no atelectasis. subcutaneous air within the left chest wall is minimal.
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mild central congestion, small effusions.
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interval increase in left lower lobe atelectasis and retrocardiac opacity which in the appropriate clinical setting may represent pneumonia. stable bilateral small pleural effusions. results were conveyed to primary team by dr on , within <num> minutes of results.
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et tube is located in the right mainstem bronchus which could be pulled back <num> cm.
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normal chest x-ray.
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persistent mild elevation of the right hemidiaphragm. mild right basilar atelectasis without definite focal consolidation.
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opacities in the lingula and right lower lobe suggesting pneumonia. follow-up chest radiographs are recommended in six to eight weeks in order to show resolution.
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no evidence of pulmonary edema or focal consolidation.
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no acute intrathoracic process.
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right mid and lower lung opacities concerning for pneumonia with mild pulmonary edema.
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linear bibasilar opacities suggestive of atelectasis, noting that infection cannot be excluded.
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as compared to the previous radiograph, no relevant change is seen. a subtle bandlike opacity in the retrocardiac lung region is likely and atelectasis and has no morphologic characteristics suggesting pneumonia. no pleural effusions. no pulmonary edema. right pectoral port-a-cath is in unchanged position.
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no acute cardiopulmonary process.
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some interval improvement is noted particularly in the right base and left upper lung zone. background significant chronic changes a chest ct is recommended at some point for further delineation
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persistent small right pleural effusion.
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ap chest compared to : moderately severe widespread pulmonary opacification, probably asymmetric pulmonary edema. the contribution of aspiration cannot be excluded. tip of the endotracheal tube is at the carina, should be withdrawn <num> cm for appropriate positioning. left lower lobe atelectasis may be a result of et tube malposition. left jugular line ends in the upper svc. no pneumothorax or mediastinal widening. dr was paged at as soon as the findings were recognized.
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no acute cardiopulmonary process.
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small left apical pneumothorax status post left chest tube removal.
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no acute cardiopulmonary abnormality. no fractures are identified. if there is continued concern, a dedicated rib series can be obtained.
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compared to chest radiographs. lungs are fully expanded and clear, aside from a calcified granuloma in the right upper lobe unchanged since. no evidence of reactivation infection. cardiomediastinal and hilar silhouettes and pleural surfaces are normal.
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mild vascular congestion and cardiomegaly. a masslike opacity seen on the same day lumbar spine ct is not appreciated on this examination. recommend dedicated chest ct for further evaluation. recommendation(s): a masslike opacity seen on the same day lumbar spine ct is not appreciated on this examination. recommend dedicated chest ct for further evaluation.
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worsening pulmonary edema; findings discussed with at am on by over the phone.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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persistent elevation of the right hemidiaphragm without acute cardiopulmonary process seen.
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et tube tip is <num> cm above the carinal. left picc line tip is in the proximal right atrium. left mediastinal shift is even more pronounced consistent most likely with left lower lobe substantial atelectasis. bilateral pleural effusions are moderate, unchanged. no pneumothorax.
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in comparison to chest radiograph, pulmonary vascular congestion and mild edema are new. bibasilar atelectasis has slightly worsened on the right and improved on the left. small left pleural effusion has apparently increased in size. no other relevant change.
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no acute intrathoracic process. hiatal hernia is minimally increased in size from the prior examination in.
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no significant change.
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no evidence of acute cardiopulmonary disease.
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no active disease.
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no acute cardiopulmonary process. no radiographic evidence of amiodarone toxicity.
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mild left basal atelectasis, otherwise unremarkable.
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no focal consolidation concerning for pneumonia.
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comparison to. decrease in extent of up right pleural effusion. decrease in severity of the interstitial opacities on the right. the left pleural effusion is stable. stable left lower lobe atelectasis. extensive apical overinflation persists.
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cardiomediastinal silhouette is within normal limits. there is again seen an area of consolidation within the right upper lobe which appears more confluent. additional opacities at the lung bases are unchanged. no pneumothoraces are seen.
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as compared to radiograph, worsening pulmonary vascular congestion is present as well as worsening bibasilar opacities. aspiration or developing aspiration pneumonia should be considered. small left pleural effusion is again demonstrated.
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mild pulmonary vascular congestion.
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subtle patchy left infrahilar opacity may be due to overlap of vascular structures although small consolidation in this region is not entirely excluded in the appropriate clinical setting.
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no acute pulmonary process identified. minimal atelectasis left base, similar to the radiographs.
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unchanged right lower lobe lung mass. known pulmonary and pleural metastases are better demonstrated on the prior ct. small left pleural effusion. interstitial opacities are more pronounced on the right, possibly reflective of asymmetric pulmonary edema but lymphangitic carcinomatosis is not excluded.
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no definite acute cardiopulmonary process.
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cardiomegaly and pulmonary engorgement, consistent with early failure.
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limited evaluation due to underlying trauma board, however no definite acute cardiopulmonary process. no acute osseous injury identified.
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low lung volumes but no evidence of acute cardiopulmonary process.
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in comparison with the study of the monitoring and support devices have been removed. the bilateral pleural effusions are well seen on the lateral view and there is continued elevation of pulmonary venous pressure. poor definition of the right heart border raises the possibility of volume loss in the middle lobe. the extensive pulmonary and pleural changes make it difficult to exclude superimposed pneumonia, as suggested by the clinical history.
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normal chest radiographic examination.
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ap chest compared to : tip of the endotracheal tube is at the level of the thoracic inlet, no less than <num> cm from the carina, with the chin elevated. it could be advanced <num> cm for more secured seating. nasogastric tube passes as far as the distal esophagus but the tip is indistinct. left subclavian line ends at the origin of the svc. right jugular line ends in the jugular vein. severe cardiomegaly is chronic. lung volumes are extremely low. edema in both upper lobes is mild, increased on the right, stable on the left. pleural effusions, if present, are not substantial. there is no pneumothorax.
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better aeration of the bilateral lungs, with improvement of bibasilar atelectasis and resolution of bilateral pleural effusions.
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no pneumothorax or displaced rib fracture. if there is strong clinical concern for rib fracture dedicated rib series may be performed.
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the et tube tip, the left picc line and the ng tube are in unchanged position. there is interval decrease in stomach distension. there is more pronounced bilateral in particular left perihilar opacities most likely concerning for pulmonary edema although other etiologies such as ards or less likely multifocal infection are a possibility
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no acute intrathoracic process.
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no acute intrathoracic process.
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no evidence of pneumonia.
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heart size and mediastinum are unchanged. bibasal atelectasis and bilateral pleural effusions are unchanged. pulmonary edema is moderate.
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in comparison with the study of , there is little change and no evidence of acute cardiopulmonary disease. mild hyperexpansion of the lungs is again seen but no evidence of acute pneumonia, vascular congestion, or pleural effusion. there is suggestion of mild impression on the right side of the lower cervical trachea, raising the possibility of thyroid enlargement.
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following removal of the rib right pigtail pleural drainage catheter, there has probably been an increase in moderate right pleural effusion obscuring much of the right lung making it difficult to say whether mild pulmonary edema has developed as well. small left pleural effusion has increased. there is no pneumothorax. cardiac silhouette is difficult to assess, probably stable and moderately enlarged. no endotracheal tube is seen. right jugular line ends in the right atrium as before. no pneumothorax.
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unchanged bilateral pulmonary edema. endotracheal tube appropriately positioned.
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endotracheal tube continues to have its tip approximately <num> to <num> cm above the carina. a left subclavian picc line is unchanged in position. pneumoperitoneum is less visible due to semi-upright technique, but persists. clinical correlation is advised. there is increasing airspace disease within the left upper and mid lung, which is asymmetric from the contralateral side. although this may represent asymmetric pulmonary edema, pneumonia should also be considered. there is persisting consolidation in the retrocardiac region which may reflect lower lobe atelectasis, although pneumonia or aspiration could also have this appearance. a left pleural catheter in the costophrenic angle is again seen with no evidence of pneumothorax. there is patchy opacity at the right base, which could reflect an area of edema, atelectasis or pneumonia. there is likely a small right effusion. no pneumothorax is appreciated. overall cardiac and mediastinal contours are likely unchanged status post median sternotomy for cabg.
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no evidence of volume overload.
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no acute cardiopulmonary abnormalities
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no acute cardiopulmonary process.
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anterior right lower lobe consolidation, most likely atelectasis. moderately severe cardiomegaly.
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relatively unchanged appearance of the chest with bilateral calcified pleural plaques and slightly hazy opacities with increased interstitial markings at the lung bases suggestive of chronic interstitial lung disease, better characterized on the previous ct exam. no new focal consolidation.
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no significant interval change in small bilateral pleural effusions bibasilar subsegmental atelectasis. no pneumothorax with right apical chest tube in place.
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resolving left lower lobe pneumonia.
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status post left pleural pigtail catheter placement with improved left pleural effusion and no pneumothorax; persisting residual retrocardiac atelectasis.
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no acute cardiopulmonary process.
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no previous images. low lung volumes, but no evidence of acute pneumonia, vascular congestion, or pleural effusion. there is mild blunting of the left costophrenic angle.
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interval decrease since in right-sided opacity with significant decrease in right pleural effusion, with small to moderate pleural effusion with overlying atelectasis residual right mid lung opacity. streaky left base opacity significantly decreased from prior radiograph and demonstrates improved aeration.
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resolution of left lower lobe pneumonia with residual left lower lobe bronchial wall thickening.
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endotracheal tube <num> cm from the carina. recommend advancing approximately <num> cm for appropriate position. improving pulmonary edema. mild edema persists. results were discussed with dr at on via telephone by dr at the time the findings were discovered.
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no acute cardiopulmonary process.
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no evidence of acute intrathoracic process.